Cellular and Molecular Biology Letters 
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This is an RSS file. You can use it to subscribe to this data in your favourite RSS reader or to display this data on your own website or blog.CircZNF609 regulates pulmonary fibrosis via miR-145-5p/KLF4 axis and its translation function
CONCLUSIONS: Our data indicate that circZNF609 is a critical regulator of fibroblast activation and silica-induced lung fibrosis. The circZNF609 and its derived peptides may represent novel promising targets for the treatment of pulmonary fibrosis.PMID:38105235 | PMC:PMC10726587 | DOI:10.1186/s11658-023-00518-w (Source: Cellular and Molecular Biology Letters)
Source: Cellular and Molecular Biology Letters - December 17, 2023 Category: Biochemistry Authors: Wenqing Sun Siyun Zhou Lan Peng Yi Liu Demin Cheng Yue Wang Chunhui Ni Source Type: research
CircZNF609 regulates pulmonary fibrosis via miR-145-5p/KLF4 axis and its translation function
CONCLUSIONS: Our data indicate that circZNF609 is a critical regulator of fibroblast activation and silica-induced lung fibrosis. The circZNF609 and its derived peptides may represent novel promising targets for the treatment of pulmonary fibrosis.PMID:38105235 | PMC:PMC10726587 | DOI:10.1186/s11658-023-00518-w (Source: Cellular and Molecular Biology Letters)
Source: Cellular and Molecular Biology Letters - December 17, 2023 Category: Biochemistry Authors: Wenqing Sun Siyun Zhou Lan Peng Yi Liu Demin Cheng Yue Wang Chunhui Ni Source Type: research
CircZNF609 regulates pulmonary fibrosis via miR-145-5p/KLF4 axis and its translation function
CONCLUSIONS: Our data indicate that circZNF609 is a critical regulator of fibroblast activation and silica-induced lung fibrosis. The circZNF609 and its derived peptides may represent novel promising targets for the treatment of pulmonary fibrosis.PMID:38105235 | PMC:PMC10726587 | DOI:10.1186/s11658-023-00518-w (Source: Cellular and Molecular Biology Letters)
Source: Cellular and Molecular Biology Letters - December 17, 2023 Category: Biochemistry Authors: Wenqing Sun Siyun Zhou Lan Peng Yi Liu Demin Cheng Yue Wang Chunhui Ni Source Type: research
CircZNF609 regulates pulmonary fibrosis via miR-145-5p/KLF4 axis and its translation function
CONCLUSIONS: Our data indicate that circZNF609 is a critical regulator of fibroblast activation and silica-induced lung fibrosis. The circZNF609 and its derived peptides may represent novel promising targets for the treatment of pulmonary fibrosis.PMID:38105235 | PMC:PMC10726587 | DOI:10.1186/s11658-023-00518-w (Source: Cellular and Molecular Biology Letters)
Source: Cellular and Molecular Biology Letters - December 17, 2023 Category: Biochemistry Authors: Wenqing Sun Siyun Zhou Lan Peng Yi Liu Demin Cheng Yue Wang Chunhui Ni Source Type: research
CircZNF609 regulates pulmonary fibrosis via miR-145-5p/KLF4 axis and its translation function
CONCLUSIONS: Our data indicate that circZNF609 is a critical regulator of fibroblast activation and silica-induced lung fibrosis. The circZNF609 and its derived peptides may represent novel promising targets for the treatment of pulmonary fibrosis.PMID:38105235 | PMC:PMC10726587 | DOI:10.1186/s11658-023-00518-w (Source: Cellular and Molecular Biology Letters)
Source: Cellular and Molecular Biology Letters - December 17, 2023 Category: Biochemistry Authors: Wenqing Sun Siyun Zhou Lan Peng Yi Liu Demin Cheng Yue Wang Chunhui Ni Source Type: research
CircZNF609 regulates pulmonary fibrosis via miR-145-5p/KLF4 axis and its translation function
CONCLUSIONS: Our data indicate that circZNF609 is a critical regulator of fibroblast activation and silica-induced lung fibrosis. The circZNF609 and its derived peptides may represent novel promising targets for the treatment of pulmonary fibrosis.PMID:38105235 | PMC:PMC10726587 | DOI:10.1186/s11658-023-00518-w (Source: Cellular and Molecular Biology Letters)
Source: Cellular and Molecular Biology Letters - December 17, 2023 Category: Biochemistry Authors: Wenqing Sun Siyun Zhou Lan Peng Yi Liu Demin Cheng Yue Wang Chunhui Ni Source Type: research
CircZNF609 regulates pulmonary fibrosis via miR-145-5p/KLF4 axis and its translation function
CONCLUSIONS: Our data indicate that circZNF609 is a critical regulator of fibroblast activation and silica-induced lung fibrosis. The circZNF609 and its derived peptides may represent novel promising targets for the treatment of pulmonary fibrosis.PMID:38105235 | PMC:PMC10726587 | DOI:10.1186/s11658-023-00518-w (Source: Cellular and Molecular Biology Letters)
Source: Cellular and Molecular Biology Letters - December 17, 2023 Category: Biochemistry Authors: Wenqing Sun Siyun Zhou Lan Peng Yi Liu Demin Cheng Yue Wang Chunhui Ni Source Type: research
CircZNF609 regulates pulmonary fibrosis via miR-145-5p/KLF4 axis and its translation function
CONCLUSIONS: Our data indicate that circZNF609 is a critical regulator of fibroblast activation and silica-induced lung fibrosis. The circZNF609 and its derived peptides may represent novel promising targets for the treatment of pulmonary fibrosis.PMID:38105235 | PMC:PMC10726587 | DOI:10.1186/s11658-023-00518-w (Source: Cellular and Molecular Biology Letters)
Source: Cellular and Molecular Biology Letters - December 17, 2023 Category: Biochemistry Authors: Wenqing Sun Siyun Zhou Lan Peng Yi Liu Demin Cheng Yue Wang Chunhui Ni Source Type: research
CircZNF609 regulates pulmonary fibrosis via miR-145-5p/KLF4 axis and its translation function
CONCLUSIONS: Our data indicate that circZNF609 is a critical regulator of fibroblast activation and silica-induced lung fibrosis. The circZNF609 and its derived peptides may represent novel promising targets for the treatment of pulmonary fibrosis.PMID:38105235 | PMC:PMC10726587 | DOI:10.1186/s11658-023-00518-w (Source: Cellular and Molecular Biology Letters)
Source: Cellular and Molecular Biology Letters - December 17, 2023 Category: Biochemistry Authors: Wenqing Sun Siyun Zhou Lan Peng Yi Liu Demin Cheng Yue Wang Chunhui Ni Source Type: research
CircZNF609 regulates pulmonary fibrosis via miR-145-5p/KLF4 axis and its translation function
CONCLUSIONS: Our data indicate that circZNF609 is a critical regulator of fibroblast activation and silica-induced lung fibrosis. The circZNF609 and its derived peptides may represent novel promising targets for the treatment of pulmonary fibrosis.PMID:38105235 | PMC:PMC10726587 | DOI:10.1186/s11658-023-00518-w (Source: Cellular and Molecular Biology Letters)
Source: Cellular and Molecular Biology Letters - December 17, 2023 Category: Biochemistry Authors: Wenqing Sun Siyun Zhou Lan Peng Yi Liu Demin Cheng Yue Wang Chunhui Ni Source Type: research
CircZNF609 regulates pulmonary fibrosis via miR-145-5p/KLF4 axis and its translation function
CONCLUSIONS: Our data indicate that circZNF609 is a critical regulator of fibroblast activation and silica-induced lung fibrosis. The circZNF609 and its derived peptides may represent novel promising targets for the treatment of pulmonary fibrosis.PMID:38105235 | DOI:10.1186/s11658-023-00518-w (Source: Cellular and Molecular Biology Letters)
Source: Cellular and Molecular Biology Letters - December 17, 2023 Category: Biochemistry Authors: Wenqing Sun Siyun Zhou Lan Peng Yi Liu Demin Cheng Yue Wang Chunhui Ni Source Type: research
Molecular mechanism and therapeutic potential of HDAC9 in intervertebral disc degeneration
CONCLUSION: Our results suggest that HDAC9 plays an important role in the development and progression of IVDD. It might be required to protect NP cells against the loss of cell viability and apoptosis by inhibiting RUNX3 acetylation and expression during IVDD. Together, our findings suggest that HDAC9 may be a potential therapeutic target in IVDD.PMID:38093179 | PMC:PMC10717711 | DOI:10.1186/s11658-023-00517-x (Source: Cellular and Molecular Biology Letters)
Source: Cellular and Molecular Biology Letters - December 13, 2023 Category: Biochemistry Authors: Ming Lei Hui Lin Deyao Shi Pan Hong Hui Song Bomansaan Herman Zhiwei Liao Cao Yang Source Type: research
Molecular mechanism and therapeutic potential of HDAC9 in intervertebral disc degeneration
CONCLUSION: Our results suggest that HDAC9 plays an important role in the development and progression of IVDD. It might be required to protect NP cells against the loss of cell viability and apoptosis by inhibiting RUNX3 acetylation and expression during IVDD. Together, our findings suggest that HDAC9 may be a potential therapeutic target in IVDD.PMID:38093179 | PMC:PMC10717711 | DOI:10.1186/s11658-023-00517-x (Source: Cellular and Molecular Biology Letters)
Source: Cellular and Molecular Biology Letters - December 13, 2023 Category: Biochemistry Authors: Ming Lei Hui Lin Deyao Shi Pan Hong Hui Song Bomansaan Herman Zhiwei Liao Cao Yang Source Type: research
Molecular mechanism and therapeutic potential of HDAC9 in intervertebral disc degeneration
CONCLUSION: Our results suggest that HDAC9 plays an important role in the development and progression of IVDD. It might be required to protect NP cells against the loss of cell viability and apoptosis by inhibiting RUNX3 acetylation and expression during IVDD. Together, our findings suggest that HDAC9 may be a potential therapeutic target in IVDD.PMID:38093179 | PMC:PMC10717711 | DOI:10.1186/s11658-023-00517-x (Source: Cellular and Molecular Biology Letters)
Source: Cellular and Molecular Biology Letters - December 13, 2023 Category: Biochemistry Authors: Ming Lei Hui Lin Deyao Shi Pan Hong Hui Song Bomansaan Herman Zhiwei Liao Cao Yang Source Type: research
Molecular mechanism and therapeutic potential of HDAC9 in intervertebral disc degeneration
CONCLUSION: Our results suggest that HDAC9 plays an important role in the development and progression of IVDD. It might be required to protect NP cells against the loss of cell viability and apoptosis by inhibiting RUNX3 acetylation and expression during IVDD. Together, our findings suggest that HDAC9 may be a potential therapeutic target in IVDD.PMID:38093179 | DOI:10.1186/s11658-023-00517-x (Source: Cellular and Molecular Biology Letters)
Source: Cellular and Molecular Biology Letters - December 13, 2023 Category: Biochemistry Authors: Ming Lei Hui Lin Deyao Shi Pan Hong Hui Song Bomansaan Herman Zhiwei Liao Cao Yang Source Type: research
