Adiponectin improves endometrial receptivity in rats with polycystic ovary syndrome by upregulating the PPAR α/HOXA10 pathway
CONCLUSION: APN can improve endometrial receptivity in PCOS rats by upregulating the PARα/HOXA10 pathway.PMID:38501415 | PMC:PMC10954532 | DOI:10.12122/j.issn.1673-4254.2024.02.12 (Source: Journal of Southern Medical University)
Source: Journal of Southern Medical University - March 19, 2024 Category: Universities & Medical Training Authors: J Wang W Yang J Liu J Shi P Xiao M Li Source Type: research
Comparative Effectiveness of Antidiabetic Drugs as an Additional Therapy to Metformin in Women with Polycystic Ovary Syndrome: A Systematic Review of Metabolic Approaches
CONCLUSION: Since PCOS is a metabolic disorder, choosing the best combination of antidiabetic drugs in the clinical course of PCOS patients will be very important. Today, it seems that we need a new metabolic approach for better treatment of the metabolic aspects of these patients.PMID:38500709 | PMC:PMC10948218 | DOI:10.1155/2024/9900213 (Source: International Journal of Endocrinology)
Source: International Journal of Endocrinology - March 19, 2024 Category: Endocrinology Authors: Maryam Heidarpour Mehrzad Mojarad Sadegh Mazaheri-Tehrani Ali Kachuei Arash Najimi Davood Shafie Hassan Rezvanian Source Type: research
Adiponectin improves endometrial receptivity in rats with polycystic ovary syndrome by upregulating the PPAR α/HOXA10 pathway
CONCLUSION: APN can improve endometrial receptivity in PCOS rats by upregulating the PARα/HOXA10 pathway.PMID:38501415 | PMC:PMC10954532 | DOI:10.12122/j.issn.1673-4254.2024.02.12 (Source: Journal of Southern Medical University)
Source: Journal of Southern Medical University - March 19, 2024 Category: Universities & Medical Training Authors: J Wang W Yang J Liu J Shi P Xiao M Li Source Type: research
Adiponectin improves endometrial receptivity in rats with polycystic ovary syndrome by upregulating the PPAR α/HOXA10 pathway
CONCLUSION: APN can improve endometrial receptivity in PCOS rats by upregulating the PARα/HOXA10 pathway.PMID:38501415 | PMC:PMC10954532 | DOI:10.12122/j.issn.1673-4254.2024.02.12 (Source: Journal of Southern Medical University)
Source: Journal of Southern Medical University - March 19, 2024 Category: Universities & Medical Training Authors: J Wang W Yang J Liu J Shi P Xiao M Li Source Type: research
Adiponectin improves endometrial receptivity in rats with polycystic ovary syndrome by upregulating the PPAR α/HOXA10 pathway
CONCLUSION: APN can improve endometrial receptivity in PCOS rats by upregulating the PARα/HOXA10 pathway.PMID:38501415 | PMC:PMC10954532 | DOI:10.12122/j.issn.1673-4254.2024.02.12 (Source: Journal of Southern Medical University)
Source: Journal of Southern Medical University - March 19, 2024 Category: Universities & Medical Training Authors: J Wang W Yang J Liu J Shi P Xiao M Li Source Type: research
Adiponectin improves endometrial receptivity in rats with polycystic ovary syndrome by upregulating the PPAR α/HOXA10 pathway
CONCLUSION: APN can improve endometrial receptivity in PCOS rats by upregulating the PARα/HOXA10 pathway.PMID:38501415 | PMC:PMC10954532 | DOI:10.12122/j.issn.1673-4254.2024.02.12 (Source: Journal of Southern Medical University)
Source: Journal of Southern Medical University - March 19, 2024 Category: Universities & Medical Training Authors: J Wang W Yang J Liu J Shi P Xiao M Li Source Type: research
Adiponectin improves endometrial receptivity in rats with polycystic ovary syndrome by upregulating the PPAR α/HOXA10 pathway
CONCLUSION: APN can improve endometrial receptivity in PCOS rats by upregulating the PARα/HOXA10 pathway.PMID:38501415 | PMC:PMC10954532 | DOI:10.12122/j.issn.1673-4254.2024.02.12 (Source: Journal of Southern Medical University)
Source: Journal of Southern Medical University - March 19, 2024 Category: Universities & Medical Training Authors: J Wang W Yang J Liu J Shi P Xiao M Li Source Type: research
Adiponectin improves endometrial receptivity in rats with polycystic ovary syndrome by upregulating the PPAR α/HOXA10 pathway
CONCLUSION: APN can improve endometrial receptivity in PCOS rats by upregulating the PARα/HOXA10 pathway.PMID:38501415 | PMC:PMC10954532 | DOI:10.12122/j.issn.1673-4254.2024.02.12 (Source: Journal of Southern Medical University)
Source: Journal of Southern Medical University - March 19, 2024 Category: Universities & Medical Training Authors: J Wang W Yang J Liu J Shi P Xiao M Li Source Type: research
Adiponectin improves endometrial receptivity in rats with polycystic ovary syndrome by upregulating the PPAR α/HOXA10 pathway
CONCLUSION: APN can improve endometrial receptivity in PCOS rats by upregulating the PARα/HOXA10 pathway.PMID:38501415 | PMC:PMC10954532 | DOI:10.12122/j.issn.1673-4254.2024.02.12 (Source: Journal of Southern Medical University)
Source: Journal of Southern Medical University - March 19, 2024 Category: Universities & Medical Training Authors: J Wang W Yang J Liu J Shi P Xiao M Li Source Type: research
Adiponectin improves endometrial receptivity in rats with polycystic ovary syndrome by upregulating the PPAR α/HOXA10 pathway
CONCLUSION: APN can improve endometrial receptivity in PCOS rats by upregulating the PARα/HOXA10 pathway.PMID:38501415 | PMC:PMC10954532 | DOI:10.12122/j.issn.1673-4254.2024.02.12 (Source: Journal of Southern Medical University)
Source: Journal of Southern Medical University - March 19, 2024 Category: Universities & Medical Training Authors: J Wang W Yang J Liu J Shi P Xiao M Li Source Type: research
Adiponectin improves endometrial receptivity in rats with polycystic ovary syndrome by upregulating the PPAR α/HOXA10 pathway
CONCLUSION: APN can improve endometrial receptivity in PCOS rats by upregulating the PARα/HOXA10 pathway.PMID:38501415 | PMC:PMC10954532 | DOI:10.12122/j.issn.1673-4254.2024.02.12 (Source: Journal of Southern Medical University)
Source: Journal of Southern Medical University - March 19, 2024 Category: Universities & Medical Training Authors: J Wang W Yang J Liu J Shi P Xiao M Li Source Type: research
Adiponectin improves endometrial receptivity in rats with polycystic ovary syndrome by upregulating the PPAR α/HOXA10 pathway
CONCLUSION: APN can improve endometrial receptivity in PCOS rats by upregulating the PARα/HOXA10 pathway.PMID:38501415 | PMC:PMC10954532 | DOI:10.12122/j.issn.1673-4254.2024.02.12 (Source: Journal of Southern Medical University)
Source: Journal of Southern Medical University - March 19, 2024 Category: Universities & Medical Training Authors: J Wang W Yang J Liu J Shi P Xiao M Li Source Type: research
Adiponectin improves endometrial receptivity in rats with polycystic ovary syndrome by upregulating the PPAR α/HOXA10 pathway
CONCLUSION: APN can improve endometrial receptivity in PCOS rats by upregulating the PARα/HOXA10 pathway.PMID:38501415 | PMC:PMC10954532 | DOI:10.12122/j.issn.1673-4254.2024.02.12 (Source: Journal of Southern Medical University)
Source: Journal of Southern Medical University - March 19, 2024 Category: Universities & Medical Training Authors: J Wang W Yang J Liu J Shi P Xiao M Li Source Type: research
Adiponectin improves endometrial receptivity in rats with polycystic ovary syndrome by upregulating the PPAR α/HOXA10 pathway
CONCLUSION: APN can improve endometrial receptivity in PCOS rats by upregulating the PARα/HOXA10 pathway.PMID:38501415 | PMC:PMC10954532 | DOI:10.12122/j.issn.1673-4254.2024.02.12 (Source: Journal of Southern Medical University)
Source: Journal of Southern Medical University - March 19, 2024 Category: Universities & Medical Training Authors: J Wang W Yang J Liu J Shi P Xiao M Li Source Type: research
Adiponectin improves endometrial receptivity in rats with polycystic ovary syndrome by upregulating the PPAR α/HOXA10 pathway
CONCLUSION: APN can improve endometrial receptivity in PCOS rats by upregulating the PARα/HOXA10 pathway.PMID:38501415 | PMC:PMC10954532 | DOI:10.12122/j.issn.1673-4254.2024.02.12 (Source: Journal of Southern Medical University)
Source: Journal of Southern Medical University - March 19, 2024 Category: Universities & Medical Training Authors: J Wang W Yang J Liu J Shi P Xiao M Li Source Type: research
