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Mitochondrial Calcium Uniporter (MCU) is Involved in an Ischemic Postconditioning Effect Against Ischemic Reperfusion Brain Injury in Mice
Cell Mol Neurobiol. 2024 Apr 3;44(1):32. doi: 10.1007/s10571-024-01464-7.ABSTRACTThe phenomenon of ischemic postconditioning (PostC) is known to be neuroprotective against ischemic reperfusion (I/R) injury. One of the key processes in PostC is the opening of the mitochondrial ATP-dependent potassium (mito-KATP) channel and depolarization of the mitochondrial membrane, triggering the release of calcium ions from mitochondria through low-conductance opening of the mitochondrial permeability transition pore. Mitochondrial calcium uniporter (MCU) is known as a highly sensitive transporter for the uptake of Ca2+ present on the ...
Source: Cellular and Molecular Neurobiology - April 3, 2024 Category: Cytology Authors: Hiromitsu Sasaki Ichiro Nakagawa Takanori Furuta Shohei Yokoyama Yudai Morisaki Yasuhiko Saito Hiroyuki Nakase Source Type: research
Mitochondrial Calcium Uniporter (MCU) is Involved in an Ischemic Postconditioning Effect Against Ischemic Reperfusion Brain Injury in Mice
Cell Mol Neurobiol. 2024 Apr 3;44(1):32. doi: 10.1007/s10571-024-01464-7.ABSTRACTThe phenomenon of ischemic postconditioning (PostC) is known to be neuroprotective against ischemic reperfusion (I/R) injury. One of the key processes in PostC is the opening of the mitochondrial ATP-dependent potassium (mito-KATP) channel and depolarization of the mitochondrial membrane, triggering the release of calcium ions from mitochondria through low-conductance opening of the mitochondrial permeability transition pore. Mitochondrial calcium uniporter (MCU) is known as a highly sensitive transporter for the uptake of Ca2+ present on the ...
Source: Cellular and Molecular Neurobiology - April 3, 2024 Category: Cytology Authors: Hiromitsu Sasaki Ichiro Nakagawa Takanori Furuta Shohei Yokoyama Yudai Morisaki Yasuhiko Saito Hiroyuki Nakase Source Type: research
Mitochondrial Calcium Uniporter (MCU) is Involved in an Ischemic Postconditioning Effect Against Ischemic Reperfusion Brain Injury in Mice
Cell Mol Neurobiol. 2024 Apr 3;44(1):32. doi: 10.1007/s10571-024-01464-7.ABSTRACTThe phenomenon of ischemic postconditioning (PostC) is known to be neuroprotective against ischemic reperfusion (I/R) injury. One of the key processes in PostC is the opening of the mitochondrial ATP-dependent potassium (mito-KATP) channel and depolarization of the mitochondrial membrane, triggering the release of calcium ions from mitochondria through low-conductance opening of the mitochondrial permeability transition pore. Mitochondrial calcium uniporter (MCU) is known as a highly sensitive transporter for the uptake of Ca2+ present on the ...
Source: Cellular and Molecular Neurobiology - April 3, 2024 Category: Cytology Authors: Hiromitsu Sasaki Ichiro Nakagawa Takanori Furuta Shohei Yokoyama Yudai Morisaki Yasuhiko Saito Hiroyuki Nakase Source Type: research
Mitochondrial Calcium Uniporter (MCU) is Involved in an Ischemic Postconditioning Effect Against Ischemic Reperfusion Brain Injury in Mice
Cell Mol Neurobiol. 2024 Apr 3;44(1):32. doi: 10.1007/s10571-024-01464-7.ABSTRACTThe phenomenon of ischemic postconditioning (PostC) is known to be neuroprotective against ischemic reperfusion (I/R) injury. One of the key processes in PostC is the opening of the mitochondrial ATP-dependent potassium (mito-KATP) channel and depolarization of the mitochondrial membrane, triggering the release of calcium ions from mitochondria through low-conductance opening of the mitochondrial permeability transition pore. Mitochondrial calcium uniporter (MCU) is known as a highly sensitive transporter for the uptake of Ca2+ present on the ...
Source: Cellular and Molecular Neurobiology - April 3, 2024 Category: Cytology Authors: Hiromitsu Sasaki Ichiro Nakagawa Takanori Furuta Shohei Yokoyama Yudai Morisaki Yasuhiko Saito Hiroyuki Nakase Source Type: research
Mitochondrial Calcium Uniporter (MCU) is Involved in an Ischemic Postconditioning Effect Against Ischemic Reperfusion Brain Injury in Mice
Cell Mol Neurobiol. 2024 Apr 3;44(1):32. doi: 10.1007/s10571-024-01464-7.ABSTRACTThe phenomenon of ischemic postconditioning (PostC) is known to be neuroprotective against ischemic reperfusion (I/R) injury. One of the key processes in PostC is the opening of the mitochondrial ATP-dependent potassium (mito-KATP) channel and depolarization of the mitochondrial membrane, triggering the release of calcium ions from mitochondria through low-conductance opening of the mitochondrial permeability transition pore. Mitochondrial calcium uniporter (MCU) is known as a highly sensitive transporter for the uptake of Ca2+ present on the ...
Source: Cellular and Molecular Neurobiology - April 3, 2024 Category: Cytology Authors: Hiromitsu Sasaki Ichiro Nakagawa Takanori Furuta Shohei Yokoyama Yudai Morisaki Yasuhiko Saito Hiroyuki Nakase Source Type: research
Mitochondrial Calcium Uniporter (MCU) is Involved in an Ischemic Postconditioning Effect Against Ischemic Reperfusion Brain Injury in Mice
Cell Mol Neurobiol. 2024 Apr 3;44(1):32. doi: 10.1007/s10571-024-01464-7.ABSTRACTThe phenomenon of ischemic postconditioning (PostC) is known to be neuroprotective against ischemic reperfusion (I/R) injury. One of the key processes in PostC is the opening of the mitochondrial ATP-dependent potassium (mito-KATP) channel and depolarization of the mitochondrial membrane, triggering the release of calcium ions from mitochondria through low-conductance opening of the mitochondrial permeability transition pore. Mitochondrial calcium uniporter (MCU) is known as a highly sensitive transporter for the uptake of Ca2+ present on the ...
Source: Cellular and Molecular Neurobiology - April 3, 2024 Category: Cytology Authors: Hiromitsu Sasaki Ichiro Nakagawa Takanori Furuta Shohei Yokoyama Yudai Morisaki Yasuhiko Saito Hiroyuki Nakase Source Type: research
Mitochondrial Calcium Uniporter (MCU) is Involved in an Ischemic Postconditioning Effect Against Ischemic Reperfusion Brain Injury in Mice
Cell Mol Neurobiol. 2024 Apr 3;44(1):32. doi: 10.1007/s10571-024-01464-7.ABSTRACTThe phenomenon of ischemic postconditioning (PostC) is known to be neuroprotective against ischemic reperfusion (I/R) injury. One of the key processes in PostC is the opening of the mitochondrial ATP-dependent potassium (mito-KATP) channel and depolarization of the mitochondrial membrane, triggering the release of calcium ions from mitochondria through low-conductance opening of the mitochondrial permeability transition pore. Mitochondrial calcium uniporter (MCU) is known as a highly sensitive transporter for the uptake of Ca2+ present on the ...
Source: Cellular and Molecular Neurobiology - April 3, 2024 Category: Cytology Authors: Hiromitsu Sasaki Ichiro Nakagawa Takanori Furuta Shohei Yokoyama Yudai Morisaki Yasuhiko Saito Hiroyuki Nakase Source Type: research
Relevance of Thymic Stromal Lymphopoietin on the Pathogenesis of Glioblastoma: Role of the Neutrophil
In conclusion, our results position TSLP as a possible new growth factor in GBM and indicate its modulation of the tumor microenvironment, particularly through its interaction with PMN.PMID:38557942 | PMC:PMC10984908 | DOI:10.1007/s10571-024-01462-9 (Source: Cellular and Molecular Neurobiology)
Source: Cellular and Molecular Neurobiology - April 1, 2024 Category: Cytology Authors: Alejandra Infante Cruz Juan Valentin Coronel Paula Saibene V élez Federico Remes Lenicov Juan Iturrizaga Mart ín Abelleyro Micaela Rosato Carolina Maiumi Shiromizu Marianela Candolfi M ónica Vermeulen Carolina Jancic Ezequiel Yasuda Silvia Berner Marce Source Type: research
Relevance of Thymic Stromal Lymphopoietin on the Pathogenesis of Glioblastoma: Role of the Neutrophil
In conclusion, our results position TSLP as a possible new growth factor in GBM and indicate its modulation of the tumor microenvironment, particularly through its interaction with PMN.PMID:38557942 | DOI:10.1007/s10571-024-01462-9 (Source: Cellular and Molecular Neurobiology)
Source: Cellular and Molecular Neurobiology - April 1, 2024 Category: Cytology Authors: Alejandra Infante Cruz Juan Valentin Coronel Paula Saibene V élez Federico Remes Lenicov Juan Iturrizaga Mart ín Abelleyro Micaela Rosato Carolina Maiumi Shiromizu Marianela Candolfi M ónica Vermeulen Carolina Jancic Ezequiel Yasuda Silvia Berner Marce Source Type: research
Exploring Heparan Sulfate Proteoglycans as Mediators of Human Mesenchymal Stem Cell Neurogenesis
Cell Mol Neurobiol. 2024 Mar 28;44(1):30. doi: 10.1007/s10571-024-01463-8.ABSTRACTAlzheimer's disease (AD) and traumatic brain injury (TBI) are major public health issues worldwide, with over 38 million people living with AD and approximately 48 million people (27-69 million) experiencing TBI annually. Neurodegenerative conditions are characterised by the accumulation of neurotoxic amyloid beta (Aβ) and microtubule-associated protein Tau (Tau) with current treatments focused on managing symptoms rather than addressing the underlying cause. Heparan sulfate proteoglycans (HSPGs) are a diverse family of macromolecules that i...
Source: Cellular and Molecular Neurobiology - March 28, 2024 Category: Cytology Authors: Sofia I Petersen Rachel K Okolicsanyi Larisa M Haupt Source Type: research
Exploring Heparan Sulfate Proteoglycans as Mediators of Human Mesenchymal Stem Cell Neurogenesis
Cell Mol Neurobiol. 2024 Mar 28;44(1):30. doi: 10.1007/s10571-024-01463-8.ABSTRACTAlzheimer's disease (AD) and traumatic brain injury (TBI) are major public health issues worldwide, with over 38 million people living with AD and approximately 48 million people (27-69 million) experiencing TBI annually. Neurodegenerative conditions are characterised by the accumulation of neurotoxic amyloid beta (Aβ) and microtubule-associated protein Tau (Tau) with current treatments focused on managing symptoms rather than addressing the underlying cause. Heparan sulfate proteoglycans (HSPGs) are a diverse family of macromolecules that i...
Source: Cellular and Molecular Neurobiology - March 28, 2024 Category: Cytology Authors: Sofia I Petersen Rachel K Okolicsanyi Larisa M Haupt Source Type: research
Exploring Heparan Sulfate Proteoglycans as Mediators of Human Mesenchymal Stem Cell Neurogenesis
Cell Mol Neurobiol. 2024 Mar 28;44(1):30. doi: 10.1007/s10571-024-01463-8.ABSTRACTAlzheimer's disease (AD) and traumatic brain injury (TBI) are major public health issues worldwide, with over 38 million people living with AD and approximately 48 million people (27-69 million) experiencing TBI annually. Neurodegenerative conditions are characterised by the accumulation of neurotoxic amyloid beta (Aβ) and microtubule-associated protein Tau (Tau) with current treatments focused on managing symptoms rather than addressing the underlying cause. Heparan sulfate proteoglycans (HSPGs) are a diverse family of macromolecules that i...
Source: Cellular and Molecular Neurobiology - March 28, 2024 Category: Cytology Authors: Sofia I Petersen Rachel K Okolicsanyi Larisa M Haupt Source Type: research
Exploring Heparan Sulfate Proteoglycans as Mediators of Human Mesenchymal Stem Cell Neurogenesis
Cell Mol Neurobiol. 2024 Mar 28;44(1):30. doi: 10.1007/s10571-024-01463-8.ABSTRACTAlzheimer's disease (AD) and traumatic brain injury (TBI) are major public health issues worldwide, with over 38 million people living with AD and approximately 48 million people (27-69 million) experiencing TBI annually. Neurodegenerative conditions are characterised by the accumulation of neurotoxic amyloid beta (Aβ) and microtubule-associated protein Tau (Tau) with current treatments focused on managing symptoms rather than addressing the underlying cause. Heparan sulfate proteoglycans (HSPGs) are a diverse family of macromolecules that i...
Source: Cellular and Molecular Neurobiology - March 28, 2024 Category: Cytology Authors: Sofia I Petersen Rachel K Okolicsanyi Larisa M Haupt Source Type: research
Retraction Note: The Interplay of Tau Protein and β-Amyloid: While Tauopathy Spreads More Profoundly Than Amyloidopathy, Both Processes Are Almost Equally Pathogenic
Cell Mol Neurobiol. 2024 Mar 21;44(1):29. doi: 10.1007/s10571-024-01468-3.NO ABSTRACTPMID:38512519 | DOI:10.1007/s10571-024-01468-3 (Source: Cellular and Molecular Neurobiology)
Source: Cellular and Molecular Neurobiology - March 21, 2024 Category: Cytology Authors: Mahsa Pourhamzeh Mohammad Taghi Joghataei Soraya Mehrabi Reza Ahadi Seyed Mohammad Massood Hojjati Nasrin Fazli Seyed Massood Nabavi Hossein Pakdaman Koorosh Shahpasand Source Type: research
Retraction Note: The Interplay of Tau Protein and β-Amyloid: While Tauopathy Spreads More Profoundly Than Amyloidopathy, Both Processes Are Almost Equally Pathogenic
Cell Mol Neurobiol. 2024 Mar 21;44(1):29. doi: 10.1007/s10571-024-01468-3.NO ABSTRACTPMID:38512519 | DOI:10.1007/s10571-024-01468-3 (Source: Cellular and Molecular Neurobiology)
Source: Cellular and Molecular Neurobiology - March 21, 2024 Category: Cytology Authors: Mahsa Pourhamzeh Mohammad Taghi Joghataei Soraya Mehrabi Reza Ahadi Seyed Mohammad Massood Hojjati Nasrin Fazli Seyed Massood Nabavi Hossein Pakdaman Koorosh Shahpasand Source Type: research