Inhibition of IGF2BP1 attenuates the progression of endometriosis through PTBP1
CONCLUSIONS: IGF2BP1 exhibits potential beneficial properties in the management of EMs by interacting with PTBP1, thereby highlighting IGF2BP1 as a promising therapeutic target for EMs.PMID:38563050 | DOI:10.5603/fhc.98213 (Source: Folia Histochemica et Cytobiologica)
Source: Folia Histochemica et Cytobiologica - April 2, 2024 Category: Cytology Authors: Yanlin Su Wencai Tian Li Cheng Ling Yin Xiaoxia He Xin Wei Source Type: research
Atypical myocytes: A quirky impersonator in surgical scar endometriosis!
Indian J Pathol Microbiol. 2024 Mar 25. doi: 10.4103/ijpm.ijpm_563_23. Online ahead of print.NO ABSTRACTPMID:38563708 | DOI:10.4103/ijpm.ijpm_563_23 (Source: Indian Journal of Pathology and Microbiology)
Source: Indian Journal of Pathology and Microbiology - April 2, 2024 Category: Pathology Authors: Archana Shetty Vijaya Mysorekar Source Type: research
Inhibition of IGF2BP1 attenuates the progression of endometriosis through PTBP1
CONCLUSIONS: IGF2BP1 exhibits potential beneficial properties in the management of EMs by interacting with PTBP1, thereby highlighting IGF2BP1 as a promising therapeutic target for EMs.PMID:38563050 | DOI:10.5603/fhc.98213 (Source: Folia Histochemica et Cytobiologica)
Source: Folia Histochemica et Cytobiologica - April 2, 2024 Category: Cytology Authors: Yanlin Su Wencai Tian Li Cheng Ling Yin Xiaoxia He Xin Wei Source Type: research
Atypical myocytes: A quirky impersonator in surgical scar endometriosis!
Indian J Pathol Microbiol. 2024 Mar 25. doi: 10.4103/ijpm.ijpm_563_23. Online ahead of print.NO ABSTRACTPMID:38563708 | DOI:10.4103/ijpm.ijpm_563_23 (Source: Indian Journal of Pathology and Microbiology)
Source: Indian Journal of Pathology and Microbiology - April 2, 2024 Category: Pathology Authors: Archana Shetty Vijaya Mysorekar Source Type: research
Inhibition of IGF2BP1 attenuates the progression of endometriosis through PTBP1
CONCLUSIONS: IGF2BP1 exhibits potential beneficial properties in the management of EMs by interacting with PTBP1, thereby highlighting IGF2BP1 as a promising therapeutic target for EMs.PMID:38563050 | DOI:10.5603/fhc.98213 (Source: Folia Histochemica et Cytobiologica)
Source: Folia Histochemica et Cytobiologica - April 2, 2024 Category: Cytology Authors: Yanlin Su Wencai Tian Li Cheng Ling Yin Xiaoxia He Xin Wei Source Type: research
Stress, pain, anxiety, and depression in endometriosis-Targeting glial activation and inflammation
Int Immunopharmacol. 2024 Apr 1;132:111942. doi: 10.1016/j.intimp.2024.111942. Online ahead of print.ABSTRACTEndometriosis (EM) is a gynecological inflammatory disease often accompanied by stress, chronic pelvic pain (CPP), anxiety, and depression, leading to a diminished quality of life. This review aims to discuss the relationship between systemic and local inflammatory responses in the central nervous system (CNS), focusing on glial dysfunctions (astrocytes and microglia) as in critical brain regions involved in emotion, cognition, pain processing, anxiety, and depression. The review presents that EM is connected to inc...
Source: International Immunopharmacology - April 2, 2024 Category: Allergy & Immunology Authors: Tahmineh Mokhtari Elnaz Irandoost Fatemeh Sheikhbahaei Source Type: research
Inhibition of IGF2BP1 attenuates the progression of endometriosis through PTBP1
CONCLUSIONS: IGF2BP1 exhibits potential beneficial properties in the management of EMs by interacting with PTBP1, thereby highlighting IGF2BP1 as a promising therapeutic target for EMs.PMID:38563050 | DOI:10.5603/fhc.98213 (Source: Folia Histochemica et Cytobiologica)
Source: Folia Histochemica et Cytobiologica - April 2, 2024 Category: Cytology Authors: Yanlin Su Wencai Tian Li Cheng Ling Yin Xiaoxia He Xin Wei Source Type: research
Atypical myocytes: A quirky impersonator in surgical scar endometriosis!
Indian J Pathol Microbiol. 2024 Mar 25. doi: 10.4103/ijpm.ijpm_563_23. Online ahead of print.NO ABSTRACTPMID:38563708 | DOI:10.4103/ijpm.ijpm_563_23 (Source: Indian Journal of Pathology and Microbiology)
Source: Indian Journal of Pathology and Microbiology - April 2, 2024 Category: Pathology Authors: Archana Shetty Vijaya Mysorekar Source Type: research
Inhibition of IGF2BP1 attenuates the progression of endometriosis through PTBP1
CONCLUSIONS: IGF2BP1 exhibits potential beneficial properties in the management of EMs by interacting with PTBP1, thereby highlighting IGF2BP1 as a promising therapeutic target for EMs.PMID:38563050 | DOI:10.5603/fhc.98213 (Source: Folia Histochemica et Cytobiologica)
Source: Folia Histochemica et Cytobiologica - April 2, 2024 Category: Cytology Authors: Yanlin Su Wencai Tian Li Cheng Ling Yin Xiaoxia He Xin Wei Source Type: research
Stress, pain, anxiety, and depression in endometriosis-Targeting glial activation and inflammation
Int Immunopharmacol. 2024 Apr 1;132:111942. doi: 10.1016/j.intimp.2024.111942. Online ahead of print.ABSTRACTEndometriosis (EM) is a gynecological inflammatory disease often accompanied by stress, chronic pelvic pain (CPP), anxiety, and depression, leading to a diminished quality of life. This review aims to discuss the relationship between systemic and local inflammatory responses in the central nervous system (CNS), focusing on glial dysfunctions (astrocytes and microglia) as in critical brain regions involved in emotion, cognition, pain processing, anxiety, and depression. The review presents that EM is connected to inc...
Source: International Immunopharmacology - April 2, 2024 Category: Allergy & Immunology Authors: Tahmineh Mokhtari Elnaz Irandoost Fatemeh Sheikhbahaei Source Type: research
Inhibition of IGF2BP1 attenuates the progression of endometriosis through PTBP1
CONCLUSIONS: IGF2BP1 exhibits potential beneficial properties in the management of EMs by interacting with PTBP1, thereby highlighting IGF2BP1 as a promising therapeutic target for EMs.PMID:38563050 | DOI:10.5603/fhc.98213 (Source: Folia Histochemica et Cytobiologica)
Source: Folia Histochemica et Cytobiologica - April 2, 2024 Category: Cytology Authors: Yanlin Su Wencai Tian Li Cheng Ling Yin Xiaoxia He Xin Wei Source Type: research
