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Total 1545 results found since Jan 2013.

Circ-100290 Regulates Autophagy-Mediated Angiogenesis of Human Umbilical Vein Endothelial Cells Induced by Conditioned Medium of Human Amniotic Mesenchymal Stem Cells
CONCLUSIONS: Circ-100290 promotes autophagy-mediated angiogenesis in hAMSC-CM-treated HUVECs.PMID:37272105 | DOI:10.24976/Discov.Med.202335176.39
Source: Discovery Medicine - June 5, 2023 Category: Research Authors: Leiting Yang Han Yan Yi Liang Dian Sun Shijun Lu Zichun Tang Ming Ma Ming Shen Source Type: research

FOXM1 Contributes to Chemotherapy Sensitivity in Cervical Cancer by Regulating TTK
CONCLUSIONS: FOXM1 regulated TTK and affected the therapeutic efficacy of cisplatin and paclitaxel in cervical cancer.PMID:37272088 | DOI:10.24976/Discov.Med.202335176.22
Source: Discovery Medicine - June 5, 2023 Category: Research Authors: Qing Tang Anli Xu Ying Yang Yunmei Zhang Jianan Sun Source Type: research

Circ-100290 Regulates Autophagy-Mediated Angiogenesis of Human Umbilical Vein Endothelial Cells Induced by Conditioned Medium of Human Amniotic Mesenchymal Stem Cells
CONCLUSIONS: Circ-100290 promotes autophagy-mediated angiogenesis in hAMSC-CM-treated HUVECs.PMID:37272105 | DOI:10.24976/Discov.Med.202335176.39
Source: Discovery Medicine - June 5, 2023 Category: Research Authors: Leiting Yang Han Yan Yi Liang Dian Sun Shijun Lu Zichun Tang Ming Ma Ming Shen Source Type: research

FOXM1 Contributes to Chemotherapy Sensitivity in Cervical Cancer by Regulating TTK
CONCLUSIONS: FOXM1 regulated TTK and affected the therapeutic efficacy of cisplatin and paclitaxel in cervical cancer.PMID:37272088 | DOI:10.24976/Discov.Med.202335176.22
Source: Discovery Medicine - June 5, 2023 Category: Research Authors: Qing Tang Anli Xu Ying Yang Yunmei Zhang Jianan Sun Source Type: research

Circ-100290 Regulates Autophagy-Mediated Angiogenesis of Human Umbilical Vein Endothelial Cells Induced by Conditioned Medium of Human Amniotic Mesenchymal Stem Cells
CONCLUSIONS: Circ-100290 promotes autophagy-mediated angiogenesis in hAMSC-CM-treated HUVECs.PMID:37272105 | DOI:10.24976/Discov.Med.202335176.39
Source: Discovery Medicine - June 5, 2023 Category: Research Authors: Leiting Yang Han Yan Yi Liang Dian Sun Shijun Lu Zichun Tang Ming Ma Ming Shen Source Type: research

FOXM1 Contributes to Chemotherapy Sensitivity in Cervical Cancer by Regulating TTK
CONCLUSIONS: FOXM1 regulated TTK and affected the therapeutic efficacy of cisplatin and paclitaxel in cervical cancer.PMID:37272088 | DOI:10.24976/Discov.Med.202335176.22
Source: Discovery Medicine - June 5, 2023 Category: Research Authors: Qing Tang Anli Xu Ying Yang Yunmei Zhang Jianan Sun Source Type: research

Circ-100290 Regulates Autophagy-Mediated Angiogenesis of Human Umbilical Vein Endothelial Cells Induced by Conditioned Medium of Human Amniotic Mesenchymal Stem Cells
CONCLUSIONS: Circ-100290 promotes autophagy-mediated angiogenesis in hAMSC-CM-treated HUVECs.PMID:37272105 | DOI:10.24976/Discov.Med.202335176.39
Source: Discovery Medicine - June 5, 2023 Category: Research Authors: Leiting Yang Han Yan Yi Liang Dian Sun Shijun Lu Zichun Tang Ming Ma Ming Shen Source Type: research

FOXM1 Contributes to Chemotherapy Sensitivity in Cervical Cancer by Regulating TTK
CONCLUSIONS: FOXM1 regulated TTK and affected the therapeutic efficacy of cisplatin and paclitaxel in cervical cancer.PMID:37272088 | DOI:10.24976/Discov.Med.202335176.22
Source: Discovery Medicine - June 5, 2023 Category: Research Authors: Qing Tang Anli Xu Ying Yang Yunmei Zhang Jianan Sun Source Type: research

Notch1 is a marker for < em > in situ < /em > resting osteocytes in a 3-dimensional gel culture model
CONCLUSIONS: We established "resting state" osteocytes using an in vitro 3D model. Notch1 can be a useful marker to help differentiate the functional states of osteocytes (activated vs. resting state).PMID:37227119 | DOI:10.1080/03008207.2023.2217271
Source: Connective Tissue Research - May 25, 2023 Category: Research Authors: Ying-Hui Zhou Jia-Yu Zhu Yue Guo Hao-Neng Tang Fang Wang Junaid Iqbal Hui-Xuan Wu Nan Hu Fen Xiao Ting Wang Long Li Hou-De Zhou Source Type: research