Post ‐COVID‐19 vaccination Guillain‐Barré syndrome with sensory ataxia, gaze‐evoked nystagmus, mental‐status change, and positive pathological reflex
ConclusionsThis is the first COVID-19 vaccine-related GBS complicated with such central nervous system manifestations. (Source: Clinical and Experimental Neuroimmunology)
Source: Clinical and Experimental Neuroimmunology - April 29, 2023 Category: Neurology Authors: Nanami Saso, Shuta Toru, Keiichi Iwasaru, Hiroaki Yokote, Toshiki Uchihara Tags: CASE REPORT Source Type: research

Neuropilin ‐1 (NRP1): A new marker for pathogenic autoreactive T‐helper cells in autoimmune disease
(Source: Clinical and Experimental Neuroimmunology)
Source: Clinical and Experimental Neuroimmunology - April 27, 2023 Category: Neurology Authors: Ben J. E. Raveney, Takashi Yamamura, Shinji Oki Tags: HIGHLIGHT Source Type: research

Neuropilin ‐1 (NRP1) – a new marker for pathogenic autoreactive T helper cells in autoimmune disease
(Source: Clinical and Experimental Neuroimmunology)
Source: Clinical and Experimental Neuroimmunology - April 17, 2023 Category: Neurology Authors: Ben J. E. Raveney, Takashi Yamamura, Shinji Oki Tags: HIGHLIGHT Source Type: research

Multifocal motor neuropathy with conduction block that was shown by the flexor digitorum profundus muscle innervated from the ulnar nerve
(Source: Clinical and Experimental Neuroimmunology)
Source: Clinical and Experimental Neuroimmunology - March 28, 2023 Category: Neurology Authors: Kazuyuki Saito, Hiroaki Yokote, Shuta Toru Tags: LETTER TO THE EDITOR Source Type: research

Bilateral Longitudinally Extensive Optic Perineuritis Post ‐COVID‐19 Presenting as “Idiopathic” Intracranial Hypertension: A Case Report
ConclusionThere is a growing association between demyelinating disorders and COVID-19 and COVID-19 vaccination, it is essential to recognize CSF abnormalities that are incompatible with a diagnosis of IIH, such as increased protein in our case, and may lead to an incorrect diagnosis.This article is protected by copyright. All rights reserved. (Source: Clinical and Experimental Neuroimmunology)
Source: Clinical and Experimental Neuroimmunology - March 25, 2023 Category: Neurology Authors: Mohammad Aladawi, Daniel Crespo, Renfeng Xu, Rana Zabad, Amrita ‐Amanda Vuppala Tags: CASE REPORT Source Type: research

Multifocal motor neuropathy with conduction block which demonstrated by the flexor digitorum profundus muscle innervated from the ulnar nerve
(Source: Clinical and Experimental Neuroimmunology)
Source: Clinical and Experimental Neuroimmunology - March 13, 2023 Category: Neurology Authors: Kazuyuki Saito, Hiroaki Yokote, Shuta Toru Tags: LETTER TO THE EDITOR Source Type: research

Cotard syndrome in anti ‐adenylate kinase 5 autoantibodies limbic encephalitis
(Source: Clinical and Experimental Neuroimmunology)
Source: Clinical and Experimental Neuroimmunology - March 8, 2023 Category: Neurology Authors: Aldo F. Costa, Alba Rodr íguez Tags: CLINICAL LETTER Source Type: research

Neuroinflammation and Neuroimmunology in Alzheimer's disease: The role of T ‐lymphocytes in Alzheimer's disease
AbstractAlzheimer's disease (AD) is the leading cause of dementia, with the number of patients with AD expected to double in the next quarter-century. Brain deposition of amyloid- β (Aβ) and tau proteins is a necessary but insufficient condition for AD pathogenesis. There is also growing evidence to suggest that chronic neuroinflammation due to excessive microglial activation and astrocyte dysfunction exacerbates the pathophysiology of AD, but the factors that disrupt these homeostatic processes remain unclear. Research into AD pathophysiology has shown interest in the changes in adaptive T-cells, which play a pivotal ro...
Source: Clinical and Experimental Neuroimmunology - March 7, 2023 Category: Neurology Authors: Norio Chihara, Asato Tsuji, Riki Matsumoto Tags: REVIEW ARTICLE Source Type: research

Multifactorial glial responses and their contributions to Alzheimer's disease continuum
AbstractAlzheimer's disease (AD) is the most common neurocognitive disorder. Various factors are intricately intertwined before clinical symptoms appear, although both amyloid- β peptide deposition and neurofibrillary tangle formation (i.e. pathological hallmarks of the AD brain) are established. Among such factors, glial responses have been increasingly recognized as important roles in the progression of these pathologies and viewed as one component of the AD continuum. However, the detailed molecular and cellular mechanisms of glial function underlying AD pathogenesis remain to be elucidated. Recent studies showed that ...
Source: Clinical and Experimental Neuroimmunology - February 25, 2023 Category: Neurology Authors: Masanori Hijioka, Tatsuya Manabe, Takashi Saito Tags: INVITED REVIEW Source Type: research

Cotard syndrome in anti ‐adenylate kinase 5 autoantibodies limbic encephalitis
(Source: Clinical and Experimental Neuroimmunology)
Source: Clinical and Experimental Neuroimmunology - February 17, 2023 Category: Neurology Authors: Aldo F. Costa, Alba Rodr íguez Tags: CLINICAL LETTER Source Type: research

Issue Information
(Source: Clinical and Experimental Neuroimmunology)
Source: Clinical and Experimental Neuroimmunology - February 15, 2023 Category: Neurology Tags: ISSUE INFORMATION Source Type: research

Development of clinical research on myasthenia gravis: Present and prospective view from Japan
(Source: Clinical and Experimental Neuroimmunology)
Source: Clinical and Experimental Neuroimmunology - February 15, 2023 Category: Neurology Authors: Hiroyuki Murai Tags: EDITORIAL Source Type: research

A thank you note to our reviewers
(Source: Clinical and Experimental Neuroimmunology)
Source: Clinical and Experimental Neuroimmunology - February 15, 2023 Category: Neurology Tags: ACKNOWLEDGMENT Source Type: research

The 34th Annual Meeting of the Japanese Society for Neuroimmunology (JSNI)
(Source: Clinical and Experimental Neuroimmunology)
Source: Clinical and Experimental Neuroimmunology - February 15, 2023 Category: Neurology Tags: ABSTRACT Source Type: research

Multifactorial glial responses and their contributions to AD continuum
AbstractAlzheimer's disease (AD) is the most common neurocognitive disorder. Various factors are intricately intertwined before clinical symptoms appear although both amyloid- β peptide (Aβ) deposition and neurofibrillary tangle formation (i.e., pathological hallmarks of the AD brain) are established. Among such factors, glial responses have been increasingly recognized as important roles in the progression of these pathologies and viewed as one component of the AD continuum. However, the detailed molecular and cellular mechanisms of glial function underlying AD pathogenesis remain to be elucidated. Recent studies reveal...
Source: Clinical and Experimental Neuroimmunology - February 10, 2023 Category: Neurology Authors: Masanori Hijioka, Tatsuya Manabe, Takashi Saito Tags: INVITED REVIEW Source Type: research