Cyclin ‐dependent kinase subunit2 (CKS2) promotes malignant phenotypes and epithelial‐mesenchymal transition‐like process in glioma by activating TGFβ/SMAD signaling
ConclusionsWe identified CKS2 as a critical contributor to the gliomagenesis, which might provide a novel therapeutic target for inhibiting the spread and infiltration of glioma.
Source: Cancer Medicine - Category: Cancer & Oncology Authors: Fan Feng,
Zongqing Zhao,
Xuechang Cai,
Xueyuan Heng,
Ximeng Ma Tags: RESEARCH ARTICLE Source Type: research
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