Filtered By:
Specialty: Reproduction Medicine
This page shows you your search results in order of relevance. This is page number 16.
Order by Relevance | Date
Total 316 results found since Jan 2013.
The balance between cell survival and death in the placenta: Do neurotrophins have a role?
Syst Biol Reprod Med. 2021 Oct 6:1-10. doi: 10.1080/19396368.2021.1980132. Online ahead of print.ABSTRACTNeurotrophins (NT) are a closely related family of growth factors, which regulate the nervous system's development, maintenance, and function. Although NTs have been well studied in neuronal cells, they are also expressed in the placenta. Despite their suggested role in regulating fetoplacental development, their precise functional significance in the placenta remains elusive. NT activate two different classes of receptors. These include the Trk, tropomyosin-related kinase family of high-affinity tropomyosin-related kin...
Source: Systems Biology in Reproductive Medicine - October 7, 2021 Category: Reproduction Medicine Authors: Prachi Pathare-Ingawale Preeti Chavan-Gautam Source Type: research
Impact of growth hormone-releasing hormone (GHRH) antagonist on Decidual stromal cell growth and apoptosis in vitro
Biol Reprod. 2021 Nov 18:ioab214. doi: 10.1093/biolre/ioab214. Online ahead of print.ABSTRACTEndometrial stromal cells remodeling is critical during human pregnancy. GHRH and its functional receptor have been shown to be expressed in gynecological cancer cells and eutopic endometrial stromal cells. Recent studies have demonstrated the potential clinical uses of antagonists of GHRH as effective antitumor agents because of its directly antagonistic effect on the locally produced GHRH in gynecological tumors. However, the impact of GHRH antagonists on normal endometrial stromal cell growth remained to be elucidated. The aim o...
Source: Reproductive Biology - November 18, 2021 Category: Reproduction Medicine Authors: Hsien-Ming Wu Liang-Hsuan Chen Andrew V Schally Hong-Yuan Huang Yung-Kuei Soong Peter C K Leung Hsin-Shih Wang Source Type: research
