Optogenetic release of norepinephrine from cardiac sympathetic neurons alters mechanical and electrical function
Conclusion
This study characterizes a powerful and clinically relevant new model for studies of cardiac arrhythmias generated by increasing the activity of sympathetic nerve terminals and the resulting activation of myocyte β-adrenergic receptors.
Source: Cardiovascular Research - Category: Cardiology Authors: Wengrowski, A. M., Wang, X., Tapa, S., Posnack, N. G., Mendelowitz, D., Kay, M. W. Tags: Integrative physiology and pathophysiology Source Type: research
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