Optogenetic release of norepinephrine from cardiac sympathetic neurons alters mechanical and electrical function

Conclusion This study characterizes a powerful and clinically relevant new model for studies of cardiac arrhythmias generated by increasing the activity of sympathetic nerve terminals and the resulting activation of myocyte β-adrenergic receptors.
Source: Cardiovascular Research - Category: Cardiology Authors: Tags: Integrative physiology and pathophysiology Source Type: research