Glycolysis-Mediated Activation of v-ATPase by Nicotinamide Mononucleotide Ameliorates Lipid-Induced Cardiomyopathy by Repressing the CD36-TLR4 Axis

CONCLUSION: NMN preserves heart function during lipid overload by preventing v-ATPase disassembly.PMID:38422177 | DOI:10.1161/CIRCRESAHA.123.322910

Source: Circulation Research - February 29, 2024 Category: Cardiology Authors: Shujin Wang Yinying Han Ruimin Liu Mengqian Hou Dietbert Neumann Jun Zhang Fang Wang Yumeng Li Xueya Zhao Francesco Schianchi Chao Dai Lizhong Liu Miranda Nabben Jan F C Glatz Xin Wu Xifeng Lu Xi Li Joost J F P Luiken Source Type: research