Low-dose Cu exposure enhanced α-synuclein accumulation associates with mitochondrial impairments in mice model of Parkinson's disease

In conclusion, low-dose copper treatment altered critical proteins involved in mitochondrial, neurodevelopmental, and inflammatory responses and affected mitochondria's ROS and ATP production levels.PMID:37717680 | DOI:10.1016/j.toxlet.2023.09.004

Source: Toxicology Letters - September 17, 2023 Category: Toxicology Authors: Jie Chen Xufang Gao Chengyou Zheng Chen Zhang Peimao Li Kaiwu He Gongping Liu Xinfeng Huang Jianjun Liu Yongmei Xie Xifei Yang Source Type: research