Evo Morales: Hero or Villain?

By Jan LundiusSTOCKHOLM / ROME, Nov 20 2019 (IPS) To be president in a country like Bolivia might be like a precarious act performed by a tightrope-dancer between “the Devil and the deep blue sea”. After 23 years as Bolivia’s President, Evo Morales finally lost his foothold and ended up as political refugee in Mexico, adding his name to a long list of previous revolutionary exiles, like Augusto Sandino, Fidel Castro, and most prominently – Leon Trotsky. The last one was murdered, though the others came back, something Evo Morales has promised to do: “Sisters and brothers, I leave for Mexico, grateful for the generosity of the government of that kindred people who gave us asylum to defend our lives. It hurts to leave the country for political reasons, but I will remain vigilant. Soon I will return with greater strength and energy.”1 Morales balance act was performed between an urban, social elite, a hostile U.S. government, suspicious neighbouring countries, big landowners, industrialists, the Army, coca growers, corrupt, political allies, alleged mistresses, regional leaders, environmentalists and not the least dissatisfaction amidst loyal supporters among poor and indigenous communities, suffering inevitable frustrations over his adminstration´s inability to provide everything they hoped for. Nevertheless, a galloping inflation was checked under Morales´s regime. Foreign currency reserves grew steadily, while millions were sp...
Source: IPS Inter Press Service - Health - Category: International Medicine & Public Health Authors: Tags: Crime & Justice Development & Aid Economy & Trade Education Featured Food & Agriculture Headlines Health Human Rights Indigenous Rights Labour Latin America & the Caribbean TerraViva United Nations Source Type: news

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Publication date: Available online 27 January 2020Source: Biochimica et Biophysica Acta (BBA) - Molecular Cell ResearchAuthor(s): Yunus Akkoc, Devrim GozuacikAbstractAutophagy is a cellular stress response mechanism activation of which leads to degradation of cellular components, including proteins as well as damaged organelles in lysosomes. Defects in autophagy mechanisms were associated with several pathologies (e.g. cancer, neurodegenerative diseases, and rare genetic diseases). Therefore, autophagy regulation is under strict control. Transcriptional and post-translational mechanisms that control autophagy in cells and ...
Source: Biochimica et Biophysica Acta (BBA) Molecular Cell Research - Category: Molecular Biology Source Type: research
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A single subanesthetic dose of ketamine, an NMDA receptor (NMDAR) antagonist, produces rapid and sustained antidepressant actions in depressed patients, addressing a major unmet need for the treatment of mood disorders. Ketamine produces a rapid increase in extracellular glutamate and synaptic formation in the prefrontal cortex, but the initial cellular trigger that initiates this increase and ketamine’s behavioral actions has not been identified. To address this question, we used a combination of viral shRNA and conditional mutation to produce cell-specific knockdown or deletion of a key NMDAR subunit, GluN2B, impli...
Source: Journal of Clinical Investigation - Category: Biomedical Science Authors: Source Type: research
Hematological malignancies have long been at the forefront of the development of novel immune-based treatment strategies. The earliest successful efforts originated from the extensive body of work in the field of allogeneic hematopoietic stem cell transplantation. These efforts laid the foundation for the recent exciting era of cancer immunotherapy, which includes immune checkpoint blockade, personal neoantigen vaccines, and adoptive T cell transfer. At the heart of the specificity of these novel strategies is the recognition of target antigens presented by malignant cells to T cells. Here, we review the advances in system...
Source: Journal of Clinical Investigation - Category: Biomedical Science Authors: Source Type: research
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Source: Journal of Clinical Investigation - Category: Biomedical Science Authors: Source Type: research
Source: Journal of Clinical Investigation - Category: Biomedical Science Authors: Source Type: research
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Source: Journal of Clinical Investigation - Category: Biomedical Science Authors: Source Type: research
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Source: Journal of Clinical Investigation - Category: Biomedical Science Authors: Source Type: research
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