Follistatin-like protein 1 attenuates doxorubicin-induced cardiomyopathy by inhibiting MsrB2-mediated mitophagy

In conclusion, these results show that FSTL1 is a novel therapeutic agent against DOX-induced cardiotoxicity that improves mitochondrial function and decreases mitophagy.PMID:38696001 | DOI:10.1007/s11010-024-04955-9

Source: Molecular and Cellular Biochemistry - May 2, 2024 Category: Biochemistry Authors: Linhe Lu Yalan Shao Nisha Wang Xiang Xiong Mengen Zhai Jiayou Tang Yang Liu Jian Yang Lifang Yang Source Type: research