Hypothesis and preliminary results on the role of MUC1 and MUC2 in relationship to autism etiology

Discussion: The results show an increase of MUC2, perhaps due to genetic origin. We hypnotize a probable over expression on highly hypoglycosylated MUC1. All this prevents a regular linkage of MUC2 to MUC1, so as to form many channels in mucosa. The free MUC2 relies on themselves and joins together with hydrogen bridge bonds, leaving the highly glycosylated end parts to become the starting point for abnormal growth of the bacteria, which in turn produce opioid such as peptides, that pass into the channels of the mucosa layer.

Source: Journal of Pediatric Neurosciences - October 17, 2023 Category: Neuroscience Authors: Menicagli Roberto Limodio Marta Source Type: research