Prenatal stress induces translational disruption associated with myelination deficits

Conclusion: The deficits early in the process of maturation of myelination indicates that the reduced myelination observed at childhood equivalence in previous studies begins in fetal life. This negative programming persists into childhood potentially due to dysregulation of MBP translation processes. Expression patterns of neurosteroidogenic enzymes in the placenta at term following stress may identify at-risk fetuses that have been exposed to a stressful in utero environment.  
Source: Developmental Neuroscience - Category: Neuroscience Source Type: research