A TOR2A Gene Product: Salusin- β Contributes to Attenuated Vasodilatation of Spontaneously Hypertensive Rats

ConclusionsThese results indicated that enhanced salusin- β activity is involved in attenuated endothelium-dependent vasodilatation pathogenesis in SHR by activating NAD(P)H oxidase derived ROS generation and inhibiting eNOS activation and NO release.

Source: Cardiovascular Drugs and Therapy - May 25, 2020 Category: Cardiology Source Type: research