Adeno-Associated Virus-Mediated Knockdown of SLC16A11 Improves Glucose Tolerance and Hepatic Insulin Signaling in High Fat Diet-Fed Mice

Conclusions Inactivation of SLC16A11, which is robustly induced by HFD, improved glucose tolerance and hepatic insulin signaling, independent of body weight, but related to TAG. Additionally, SLC16A11 might mediate the health benefits of endurance exercise. [...] © Georg Thieme Verlag KG Stuttgart · New YorkArticle in Thieme eJournals: Table of contents  |  Abstract  |  Full text

Source: Experimental and Clinical Endocrinology and Diabetes - June 10, 2019 Category: Endocrinology Authors: Zhang, Tan Qi, Zhengtang Wang, Haiyan Ding, Shuzhe Tags: Article Source Type: research