Music therapy program at UCLA aims to help premature infants develop feeding skills

It ’s a Friday morning in January when Jana Gallus, 32, and Gregor Martynus, 35, peek their heads over their triplets’ set of bassinets in their Westwood apartment. Six-month-olds Ada, Kian and Nico are just waking up, kicking with excitement and beaming from ear to ear.It ’s a daily moment that Gallus and Martynus say doesn’t get old. The triplets have come a long way since July, when they were born early, at 31 weeks, with weights ranging from 2.5 to 4 pounds. For 52 days, the trio were cared for in the neonatal intensive care unit, or NICU, atUCLA Mattel Children ’s Hospital.As the UCLA team of nurses, physicians and staff monitored the triplets, the delicate newborns were met by another care team: a group of music therapists using an auditory device aimed at helping preemies develop, feed and grow.Premature infants in the NICU — especially those born before 34 weeks — struggle with oral feeding. They typically haven’t developed the reflex to suck, breathe and swallow, which inhibits their ability to gain weight.It ’s for this reason that music therapist Jenna Bollard sought to study whether a pacifier-activated lullaby device, or PAL, might help. The device is a white, rectangular object about the size of a shoebox with an electronic screen and a pacifier connected via a short cord. Its secret ingredient: t he music it plays — a personal lullaby that parents write, sing and record for their baby with the help of ...
Source: UCLA Newsroom: Health Sciences - Category: Universities & Medical Training Source Type: news

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Publication date: Available online 19 March 2019Source: SeizureAuthor(s): Ilaria Lagorio, Federico Zara, Salvatore Striano, Pasquale StrianoAbstractFamilial adult myoclonic epilepsy (FAME), also described with different acronyms (ADCME, BAFME, FEME, FCTE and others), is a high-penetrant autosomal dominant condition featuring cortical hand tremors, myoclonic jerks, and occasional/rare convulsive seizures. Prevalence is unknown since this condition is often under-recognized, but it is estimated to be less than 1/35,000. The disease usually starts in the second decade of life and has been genetically associated with at least ...
Source: Seizure - Category: Neurology Source Type: research
Abstract Obstructive sleep apnea (OSA) is associated with adverse physiologic and biochemical sequelae including systemic inflammation, oxidative stress, increased chemoreceptor reflex activity, sympathetic nervous system activation, and endothelial dysfunction. In addition, compelling data strongly implicate OSA in the development of cardiovascular disease (CVD) including myocardial infarction and stroke. Yet in clinical trials, CPAP therapy alone did not reduce cardiovascular events in intention to treat analyses. Thus, opportunity arises for adjunctive therapy, independent of treating comorbid conditions, to ta...
Source: American Journal of Respiratory and Critical Care Medicine - Category: Respiratory Medicine Authors: Tags: Am J Respir Crit Care Med Source Type: research
Obstructive sleep apnea (OSA) is the most common form of sleep disordered breathing (SDB). OSA is characterized by recurrent collapses of the upper airway during sleep and it is associated with intermittent hypoxia, sleep fragmentation, surges of sympathetic tone, and oxidative stress, finally resulting in increased cardiovascular risk and excessive daytime sleepiness. The prevalence of OSA has been reported to be 22% among men and 17% among women [1]. Women, though, are less likely to report typical OSA symptoms such as loud snoring, excessive sleepiness, and choking during sleep.
Source: Maturitas - Category: Primary Care Authors: Source Type: research
Obstructive sleep apnoea (OSA) is a common disorder in which loss of pharyngeal dilator muscle tone during sleep causes recurrent collapse of the upper airway and temporary cessation of breathing. The recurrent apnea –hypopnea episodes cause autonomic alterations, including activation of the sympathetic nervous system and an increase in serum catecholamine levels, which in turn increase both heart rate and blood pressure. Also, cortical arousals from sleep, which are brief apnea-related awakenings, and altered sleep quality result in sympathetic nerve activation in OSA patients.
Source: Maturitas - Category: Primary Care Authors: Source Type: research
Fatal Familial Insomnia (FFI) is a hereditary prion disease caused by a mutation at codon 178 of the prion-protein gene leading to a D178N substitution in the protein determining severe and selective atrophy of mediodorsal and anteroventral thalamic nuclei. FFI is characterized by physiological sleep loss, which polygraphically appears to be a slow wave sleep loss, autonomic and motor hyperactivation with peculiar episodes of oneiric stupor.Alteration of autonomic functions is a great burden for FFI patients consisting in sympathetic overactivation, dysregulation of its physiological responses and disruption of circadian rhythms.
Source: Autonomic Neuroscience: Basic and Clinical - Category: Neuroscience Authors: Tags: Review Source Type: research
Heart rate variability is now well-established as a means to assess autonomic nervous system (ANS) control by analyzing the beat-to-beat variability over time of consecutive heart beats. The R-R intervals are mathematically transformed and depicted as power spectra within different frequency domains. Low frequency reflects both sympathetic and parasympathetic activity, high frequency reflects parasympathetic activity, and total power reflects overall autonomic activity. These analyses have been applied to a wide range of clinical conditions, and investigators have studied maturation of ANS control in preterm and term infan...
Source: The Journal of Pediatrics - Category: Pediatrics Authors: Tags: The Editors' Perspectives Source Type: research
ConclusionsBMS patients showed thermal hypoesthesia within LN distribution compatible with small fibre neuropathy. The DRD2 957C>T genotype influences perception and experience of BMS pain.This article is protected by copyright. All rights reserved.
Source: European Journal of Pain - Category: Anesthesiology Authors: Tags: Original Article Source Type: research
Myotonic dystrophy type I (DM1; Online Mendelian Inheritance in Man ® ID 160900) is a hereditary multi-system disorder characterized by myopathic facies, distal muscle wasting, myotonia, bilateral cataract, endocrine and cardiac conduction abnormalities. The prevalence of DM1 in industrialized countries is 3-15/100,000. DM1 follows an autosomal dominant trait and i s caused by an expansion of a trinucleotide CTG-repeat in the 3’-untranslated region of the DMPK gene on chromosome 19 [1].
Source: Neuromuscular Disorders - Category: Neurology Authors: Source Type: research
Introduction/Purpose We demonstrated that patients with obstructive sleep apnea (OSA) have reduced muscle metaboreflex control of muscle sympathetic nerve activity (MSNA). In addition, exercise training increased muscle metaboreflex control in heart failure patients. Objective We tested the hypothesis that exercise training would increase muscle metaboreflex control of MSNA in patients with OSA. Methods Forty-one patients with OSA were randomized into the following two groups: 1) nontrained (OSANT, n = 21) and 2) trained (OSAT, n = 20). Muscle sympathetic nerve activity was assessed by microneurography technique, m...
Source: Medicine and Science in Sports and Exercise - Category: Sports Medicine Tags: CLINICAL SCIENCES Source Type: research
This study tested the hypothesis that GMM diets modulate stress-evoked sterile inflammation. Male F344 rats were assigned either GMM diets or isocaloric-nutrient matched control diet. After 4 weeks, rats were exposed to stress (intermittent tailshock-90 min) or remained in their home cages. Immediately after stressor termination, rats were sacrificed and blood and tissues were collected. Consistent with previous results, GMM diets increased stress-protective gut bacteria (fecal L. rhamnosus). Although GMM diet minimally impacted stress-induced increases in corticosterone, glucose or splenic contraction (indicative of s...
Source: Brain, Behavior, and Immunity - Category: Neurology Source Type: research
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