Filtered By:
Specialty: Neuroscience
Source: Clinical Neurophysiology
This page shows you your search results in order of date. This is page number 15.
Order by Relevance | Date
Total 347 results found since Jan 2013.
27. Botulotoxin in the treatment of spasticity – Our experience
Conclusion: Btx is an effective and safe treatment. An integral part of the treatment program for patients with spasticity must be rehabilitation, stretching, splinting, physical processes. The prerequisite is the motivation and cooperation of patients.
Source: Clinical Neurophysiology - April 7, 2014 Category: Neuroscience Authors: J. Kubik, P. Bodnárová, R. Michánková, P. Klepiš, J. Neumann Tags: Society Proceedings Source Type: research
7. Convulsive and non-convulsive seizures observed from aging mice following brian ischemia episodes
Conclusions: The early-onset seizures result from severe cerebral ischemia and brain injury. Generation of the convulsive seizures may involve deeper sub-cortical structures particularly the brainstem, and the non-convulsive EEG discharges may originate from the hippocampus. Our data may help understanding genesis of post-stroke seizures in the aging/aged population.
Source: Clinical Neurophysiology - April 7, 2014 Category: Neuroscience Authors: Liang Zhang Tags: Society Proceedings Source Type: research
Activation deficit correlates with weakness in chronic stroke: Evidence from evoked and voluntary EMG recordings
Weakness after stroke is widely observed clinically, and is reported to be the primary contributor to impaired voluntary force control (Chang et al., 2013) and to functional impairments in chronic stroke (Kamper et al., 2006). Weakness is highly correlated with the severity of initial damage to the corticospinal tracts in the acute phase (Small et al., 2013). In the course of recovery, both central and peripheral mechanisms contribute to weakness as a result of neural plasticity, adaptation, exercises and therapies.
Source: Clinical Neurophysiology - April 3, 2014 Category: Neuroscience Authors: Sheng Li, Jie Liu, Minal Bhadane, Ping Zhou, W. Zev Rymer Source Type: research
A region-based two-step P300-based brain–computer interface for patients with amyotrophic lateral sclerosis
The brain–computer interface (BCI) or brain–machine interface (BMI) is an interface technology that enables communication with others and control of the environment or of a prosthesis without any muscle movement (Wolpaw et al., 2002; Birbaumer and Cohen, 2007; Daly and Wolpaw, 2008). In this decade, the use of BCI technology has become widespread, mainly for preclinical research, due to technical and mechanical improvements, and new technology been designed to help individuals with severe neurological disabilities, especially motor difficulties such as amyotrophic lateral sclerosis (ALS), spinal cord injury (SCI), and cerebral stroke.
Source: Clinical Neurophysiology - March 26, 2014 Category: Neuroscience Authors: Shiro Ikegami, Kouji Takano, Kiyohiko Kondo, Naokatsu Saeki, Kenji Kansaku Source Type: research
Changes in diffusion tensor tractographic findings associated with constraint-induced movement therapy in young children with cerebral palsy
Constraint-induced movement therapy (CIMT) involves the application of constraint applied to the unimpaired upper limb coupled with intensive training of unimanual skills in the hemiplegic arm (Hoare et al., 2007). CIMT has been studied extensively and found to be effective in the treatment of adult hemiparetic stroke (Sirtori et al., 2009). Several randomized clinical trials performed on children with cerebral palsy (CP) also demonstrated immediate gain in the frequency of use and improved movement efficacy of the impaired upper limb (Deppe et al., 2013; Hoare et al., 2007; Rostami and Malamiri, 2012).
Source: Clinical Neurophysiology - March 20, 2014 Category: Neuroscience Authors: Jeong-Yi Kwon, Won Hyuk Chang, Hyun Jung Chang, Sook-Hee Yi, Min-Young Kim, Eun-Hye Kim, Yun-Hee Kim Source Type: research
Asymmetries in vestibular evoked myogenic potentials in chronic stroke survivors with spastic hypertonia: Evidence for a vestibulospinal role
Spastic hypertonia or “spasticity” is a frequent and often disabling sequel to hemispheric stroke (Watkins et al., 2002; Urban et al., 2010). It is a motor disorder, manifesting as a sharply lateralized muscular hypertonia on the contralesional side with exaggerated phasic and tonic stretch reflex activity (Lance, 1980). Clinically, spasticity presents as an increase in the resistance of a passive limb to externally applied joint motion and is commonly associated with deficits in both motor and functional performance (Bohannon et al., 1987; O’Dwyer et al., 1996; Watkins et al., 2002; Sommerfeld et al., 2004).
Source: Clinical Neurophysiology - March 13, 2014 Category: Neuroscience Authors: Derek M. Miller, Cliff S. Klein, Nina L. Suresh, William Z. Rymer Source Type: research
8. Evolution and recovery of motor function post-stroke
Conclusion: Intracortical disinhibition occurs in both hemispheres immediately following acute stroke that likely represent functionally relevant changes supporting the recovery process in stroke. Whilst changes in the lesioned hemisphere persisted over the period of recovery in all patients, contralesional intracortical hyperexcitability persisted in patients with cortical strokes and those with more severe baseline functional impairment, suggesting that ongoing contralesional network recruitment may be necessary for those patients who have significant disruptions to the integrity of ipsilesional motor pathways. The study...
Source: Clinical Neurophysiology - March 3, 2014 Category: Neuroscience Authors: William Huynh, Steve Vucic, Arun V. Krishnan, Cindy S.-Y. Lin, Matthew C. Kiernan Tags: Society Proceedings Source Type: research
How do the physiology and transcallosal effects of the unaffected hemisphere change during inpatient rehabilitation after stroke?
In this issue of Clinical Neurophysiology, Takechi and colleagues publish a paper entitled “Longitudinal changes of motor cortical excitability and transcallosal inhibition after subcortical stroke” that represents a long-overdue examination of changes in interhemispheric interactions during an important period for the recovery of motor function after stroke. It has been suspected that this type of recovery to neuronal damage represents a type of neuroplasticity, potentially with both adaptive and maladaptive processes.
Source: Clinical Neurophysiology - March 3, 2014 Category: Neuroscience Authors: George F. Wittenberg, Michael A. Dimyan Tags: Editorial Source Type: research
Longitudinal changes of motor cortical excitability and transcallosal inhibition after subcortical stroke
Motor circuit reorganization in the cerebral cortex is known to contribute to recovery following stroke. This reorganization can be examined by transcranial magnetic stimulation (TMS) using measures of corticospinal and intracortical excitability (Liepert et al., 2000; Shimizu et al., 2002; Liepert et al., 2005; Talelli et al., 2006; Wittenberg et al., 2007; Bütefisch et al., 2008; Manganotti et al., 2008; Swayne et al., 2008; Takeuchi et al., 2010). TMS measures such as motor threshold (MT) and recruitment curve (RC) reflect corticospinal excitability, whereas intracortical excitability is reflected by cortical silent pe...
Source: Clinical Neurophysiology - March 3, 2014 Category: Neuroscience Authors: Utako Takechi, Kaoru Matsunaga, Ryoji Nakanishi, Hiroaki Yamanaga, Nobuki Murayama, Kosuke Mafune, Sadatoshi Tsuji Source Type: research
