Activation deficit correlates with weakness in chronic stroke: Evidence from evoked and voluntary EMG recordings

Weakness after stroke is widely observed clinically, and is reported to be the primary contributor to impaired voluntary force control (Chang et al., 2013) and to functional impairments in chronic stroke (Kamper et al., 2006). Weakness is highly correlated with the severity of initial damage to the corticospinal tracts in the acute phase (Small et al., 2013). In the course of recovery, both central and peripheral mechanisms contribute to weakness as a result of neural plasticity, adaptation, exercises and therapies.

Source: Clinical Neurophysiology - April 3, 2014 Category: Neuroscience Authors: Sheng Li, Jie Liu, Minal Bhadane, Ping Zhou, W. Zev Rymer Source Type: research