Deciphering the possible role of H 2 O 2 in methylmercury-induced neurotoxicity in Xenopus laevis

ConclusionThese results supported the idea that neurodevelopment-related gene expression was regulated by oxidative stress and that apoptosis and autophagy pathways were activated by H2O2 and involved in methylmercury neurotoxicity.
Source: Molecular and Cellular Toxicology - Category: Cytology Source Type: research