Over-Expression of Inhibitor of Differentiation 2 Attenuates Post-Infarct Cardiac Fibrosis Through Inhibition of TGF- β1/Smad3/HIF-1α/IL-11 Signaling Pathway
Conclusion: In conclusion, this study demonstrated that over-expression of Id2 preserved cardiac function and ameliorated adverse cardiac remodeling, which might be a promising treatment target for cardiac fibrosis and apoptosis.
Source: Frontiers in Pharmacology - Category: Drugs & Pharmacology Source Type: research
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