Exercise preconditioning and low dose copper nanoparticles exhibits cardioprotection through targeting GSK-3β phosphorylation in ischemia/reperfusion induced myocardial infarction

ConclusionTreatment with CuNP and exercise training either alone or in combination favourably phosphorylate GSK-3β kinase pathways and further diminish oxidative stress, inflammatory cytokines, apoptosis and increase serum bioavailability of NO in the I/R insulted rats which tends to protect myocardial damage.Graphical abstract
Source: Microvascular Research - Category: Biochemistry Source Type: research