Gab1 Overexpression Attenuates Susceptibility to Ventricular Arrhythmias in Pressure Overloaded Heart Failure Mouse Hearts
ConclusionsOur study manifested that Gab1 may serve as a promising anti-arrhythmic target via inhibiting TLR4/MyD88/NF- κB signaling pathway induced by AB.
Source: Cardiovascular Drugs and Therapy - Category: Cardiology Source Type: research
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