Homocysteine modulates CXCL10/CXCR3 axis activity to induce endothelial dysfunction

In conclusion, our findings illuminated the central role of the CXCL10/CXCR3 axis in mediating Hcy-induced endothelial dysfunction, providing valuable insights for potential therapeutic strategies in managing HHcy-related cardiovascular diseases.PMID:38430022 | DOI:10.14715/cmb/2024.70.2.28

Source: Cellular and Molecular Biology - March 2, 2024 Category: Molecular Biology Authors: Yanjie Xu Yingying Xu Zuozong Yu Dingwen He Source Type: research