VD/VDR-mediated ATG16L1 activation reduces Alzheimer ’s disease-like pathology and cognitive decline
ConclusionCCE exhibits potential in safeguarding hippocampal neurons against AD-induced harm, stimulating autophagy, and suppressing inflammation. These effects may be regulated by VD/VDR/ATG16L1 signaling.
Source: Molecular and Cellular Toxicology - Category: Cytology Source Type: research
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