TMEM16A inhibits renal tubulointerstitial fibrosis via Wnt/ β-catenin signaling during hypertension nephropathy

CONCLUSION: In angiotensin II (Ang II)-induced hypertensive nephropathy, TMEM16A was identified as a key player inhibiting the detrimental changes in renal tubules, suggesting a potential avenue for mitigating renal damage in hypertensive nephropathy.PMID:38316267 | DOI:10.1016/j.cellsig.2024.111088
Source: Cellular Signalling - Category: Cytology Authors: Source Type: research