Filtered By:
Specialty: Cardiology
Procedure: Carotid Endarterectomy

This page shows you your search results in order of date. This is page number 3.

Order by Relevance | Date

Total 244 results found since Jan 2013.

Interleukin-6 Predicts Carotid Plaque Severity, Vulnerability, and Progression
CONCLUSIONS: Circulating IL-6 predicts carotid plaque severity, vulnerability, and progression. The 2.0 pg/mL cutoff could facilitate the selection of individuals that would benefit from anti-IL-6 drugs for stroke prevention.PMID:35713008 | DOI:10.1161/CIRCRESAHA.122.320877
Source: Atherosclerosis - June 17, 2022 Category: Cardiology Authors: Joseph Kamtchum-Tatuene Luca Saba Mirjam R Heldner Michiel H F Poorthuis Gert J de Borst Tatjana Rundek Stavros K Kakkos Seemant Chaturvedi Raffi Topakian Joseph F Polak Glen C Jickling Carotid Atherosclerosis and Stroke Collaboration (CASCO) Source Type: research

Lipid Droplet Protein PLIN1 Regulates Inflammatory Polarity in Human Macrophages and is Involved in Atherosclerotic Plaque Development by Promoting Stable Lipid Storage
CONCLUSION: There was a relationship between macrophage polarity, cytosolic LD size, and PLIN1/PLIN2 expression levels. PLIN2 was mainly expressed in arterial plaques in symptomatic stroke patients, and associated with the inflammatory phenotype of human macrophages, while PLIN1 expression is closely associated with plaque stability and the anti-inflammatory phenotype.PMID:35662076 | DOI:10.5551/jat.63153
Source: Journal of Atherosclerosis and Thrombosis - June 6, 2022 Category: Cardiology Authors: Kyu Yong Cho Hideaki Miyoshi Akinobu Nakamura Andrew S Greenberg Tatsuya Atsumi Source Type: research

Uric Acid Expression in Carotid Atherosclerotic Plaque and Serum Uric Acid Are Associated With Cerebrovascular Events
CONCLUSIONS: The current study supports a potential role of UA as a potential tissue participant and a systemic biomarker in the pathogenesis of carotid atherosclerosis. UA may provide a mechanistic explanation for plaque instability and subsequent ischemic cerebrovascular events.PMID:35656807 | DOI:10.1161/HYPERTENSIONAHA.122.19247
Source: Atherosclerosis - June 3, 2022 Category: Cardiology Authors: Valentina Nardi Federico Franchi Megha Prasad Erica M Fatica Mariam P Alexander Melanie C Bois Josephine Lam Ravinder J Singh Fredric B Meyer Giuseppe Lanzino Yuning Xiong Esther Lutgens Lilach O Lerman Amir Lerman Source Type: research

One-Year Outcomes After Transcarotid Artery Revascularization (TCAR) in the ROADSTER 2 TRIAL
CONCLUSIONS: In patients with high risk factors, TCAR yields high technical success with a low stroke and death rate at one year. Further comparative studies to CEA are warranted.PMID:35381327 | DOI:10.1016/j.jvs.2022.03.872
Source: Atherosclerosis - April 5, 2022 Category: Cardiology Authors: Vikram S Kashyap Kristine L So Peter A Schneider Rama Rathore Thi Pham Raghu L Motaganahalli Douglas W Massop Mazin I Foteh Hans-Henning Eckstein Jeffrey Jim Jose Ignacio Leal Lorenzo James G Melton Source Type: research

Carotid Plaque Imaging: Strategies beyond Stenosis
Ann Indian Acad Neurol. 2022 Jan-Feb;25(1):11-14. doi: 10.4103/aian.aian_483_21. Epub 2021 Aug 23.ABSTRACTAtherosclerosis at the carotid bifurcation in the neck is common and the result of turbulent hemodynamics in the bulb. Carotid bulb plaques cause disabling strokes. Carotid vessel wall imaging characterizes the atherosclerotic plaque, documents disease progression, and assesses lesion severity by plaque morphology in addition to the degree of stenosis. This in turn optimizes treatment selection for the individual patient. The aim of vessel wall imaging in patients of stroke is to distinguish stable plaques that may be ...
Source: Atherosclerosis - March 28, 2022 Category: Cardiology Authors: Darshana Sanghvi Manish Shrivastava Source Type: research

Plaque Evaluation by Ultrasound and Transcriptomics Reveals BCLAF1 as a Regulator of Smooth Muscle Cell Lipid Transdifferentiation in Atherosclerosis
CONCLUSIONS: Plaque echogenicity correlated with enrichment of distinct molecular pathways and identified BCLAF1, previously not described in atherosclerosis, as the most significant gene. Functionally, BCLAF1 seems necessary for survival and transdifferentiation of SMCs into a macrophage-like phenotype. The role of BCLAF1 in plaque vulnerability should be further evaluated.PMID:35321563 | DOI:10.1161/ATVBAHA.121.317018
Source: Arteriosclerosis, Thrombosis and Vascular Biology - March 24, 2022 Category: Cardiology Authors: Urszula Rykaczewska Quanyi Zhao Peter Saliba-Gustafsson Mariette Lengquist Malin Kronqvist Otto Bergman Zhiqiang Huang Kent Lund Katarina Waden Vila Z Pons Kenneth Caidahl Josefin Skogsberg Vladana Vukojevic Jan H N Lindeman Joy Roy G öran K Hansson Ecka Source Type: research