Why do we NOT name Occlusion MI (OMI) after an EKG finding? (In contrast to STEMI, which is named after ST Elevation)

A 40-something male with no previous cardiac disease presented with chest pain.Here is his ECG:There is no clear evidence of OMI or ischemia.  There is a tiny amount of STE in aVL, but it is NOT in the context of a tiny R-wave.  There is a tiny amount of STD in lead III, with some non-specific T-wave flattening.I am glad that Ken Grauer (below) brings up the issue of whether the presence of " T-wave in V1 taller than T-wave in V6 " is evidence for OMI.  I showed conclusively that this is a common finding in normal ECGs, though it is more common in LAD Occlusion than in norml variant STE.  Moreover,the research which appears to confirm this idea was indeed in relation to the circumflex,but they did not study Occlusion; rather, they studied asymptomatic coronary disease.  Studies of normal ECGs show that the T-wave in V1 taller than T-wave in V6 is within the normal range. The pain continued and the first high sensitivity troponin I returned at 105 ng/LAnother ECG was recorded:The ST segment in aVF has flattened a bit, revealing that there is some STD in addition to the non-specific findings in III and aVL.  This is suspicious for inferior reciprocal At this point, with continued pain, cath lab activation is indicated.  Acute chest pain that is persistent (refractory to treatment), with a significantly elevated tropoinin, is due to OMI until proven otherwise.even in the absence of ECG findings.But the cath lab was not activ...
Source: Dr. Smith's ECG Blog - Category: Cardiology Authors: Source Type: blogs