Extracellular Vesicles Derived from Intermittent Hypoxia –Treated Red Blood Cells Impair Endothelial Function Through Regulating eNOS Phosphorylation and ET-1 Expression

ConclusionsThis study reveals a novel role of REVs in endothelial dysfunction under IH and dissects the relevant mechanism involved in this process, which will help to establish a comprehensive understanding of OSA or IH-related endothelial dysfunction from a new scope.
Source: Cardiovascular Drugs and Therapy - Category: Cardiology Source Type: research