Scientific pathophysiology of intrapartum fetal hypoxemia and cardiotocography pattern recognition –realignment is a basic prerequisite!

Turner et  al1 highlight knowledge accrued since the 1960s that 75% cases of fetal hypoxemia result from contraction-induced reduction in uteroplacental perfusion by up to 60%. Following anomalies oblige focused explicit clarification by the authors. The review maintains a misconception that the chemoreflex would increase the cardiac output.1 However, with a significant fetal heart rate (FHR) deceleration intended to reduce cardiac workload and oxygen consumption, the stroke-volume could increase, but the cardiac output per minute would drop albeit preferentially redistributed to the brain.
Source: American Journal of Obstetrics and Gynecology - Category: OBGYN Authors: Tags: Letter to the Editors Source Type: research