Tafluprost promotes axon regeneration after optic nerve crush via Zn < sup > 2+ < /sup > -mTOR pathway

CONCLUSIONS: Our results suggest that Tafluprost promoted axon regeneration via regulation of the Zn2+-mTOR pathway, and provide novel research directions for glaucomatous optic nerve injury mechanisms.PMID:37832634 | DOI:10.1016/j.neuropharm.2023.109746
Source: Neuropharmacology - Category: Drugs & Pharmacology Authors: Source Type: research