SEC61G knockdown enhances the sensitivity of gastric cancer cells to etoposide through EGFR and glycolytic-mediated pathways

ConclusionsSEC61G knockdown weakened the proliferation, EGFR phosphorylation and glycolysis of gastric cancer cells, as well as increased the sensitivity of gastric cancer to etoposide, which make SEC61G a promising therapeutic target for gastric cancer treatment.

http://link.springer.com/10.1007/s13273-023-00403-9

Source: Molecular and Cellular Toxicology - October 14, 2023 Category: Cytology Source Type: research