Salidroside attenuates cerebral ischemia/reperfusion injury by regulating TSC2-induced autophagy

In conclusion, SAL reduces the inflammatory response and oxidative stress in a concentration-dependent manner, and protects against cerebral I/R injury by modulating TSC2-induced autophagy. These findings suggest SAL may prove to be a potential therapeutic agent for ischemic stroke.PMID:36374318 | DOI:10.1007/s00221-022-06493-6
Source: Cell Research - Category: Cytology Authors: Source Type: research