Calmodulin Kinase IV‐dependent CREB activation is required for neuroprotection via NMDA receptor‐PSD95 disruption

This study highlights the importance of maintaining neuronal function following ischemic injury. Future stroke research should target neurotrophic and pro‐survival signal pathways in the development of novel neuroprotective strategies. © 2013 International Society for Neurochemistry, J. Neurochem. (2013) 10.1111/jnc.12176

http://onlinelibrary.wiley.com/resolve/doi?DOI=10.1111%2Fjnc.12176

Source: Journal of Neurochemistry - January 31, 2013 Category: Neurology Authors: Karen F. S. Bell, Russell J. Bent, Saira Meese‐Tamuri, Alicia Ali, Joan P. Forder, Michelle M. Aarts Tags: Original Article Source Type: research