NR2B Phosphorylation at Tyrosine 1472 Contributes to Brain Injury in a Rodent Model of Neonatal Hypoxia-Ischemia [Basic Sciences]

Conclusions— Upregulation of NR2B phosphorylation at Y1472 after neonatal HI is involved in superoxide-mediated oxidative stress and contributes to brain injury.

http://stroke.ahajournals.org/cgi/content/short/45/10/3040?rss=1

Source: Stroke - September 22, 2014 Category: Neurology Authors: Knox, R., Brennan-Minnella, A. M., Lu, F., Yang, D., Nakazawa, T., Yamamoto, T., Swanson, R. A., Ferriero, D. M., Jiang, X. Tags: Animal models of human disease Basic Sciences Source Type: research