Schwann Cell Plasticity is Regulated by a Weakened Intrinsic Antioxidant Defense System in Acute Peripheral Nerve Injury

In conclusion, the Nrf2-antioxidant system was temporarily inactivated in injured nerves, promoting Schwann cell reprogramming and proliferation, and its functional recovery was essential for Schwann cell redifferentiation and myelination.
Source: Neuroscience - Category: Neuroscience Source Type: research