Possible involvement of tetrahydrobiopterin in the disturbance of redox homeostasis in sepsis - induced brain dysfunction.

CONCLUSION: Our results suggest that BH4 and/or ALC might protect the brain against oxidative stress induced neuronal apoptosis by restoring bioavailability of BH4 and upregulating of BH4 synthetic enzymes in the brain during sepsis. PMID: 29428597 [PubMed - as supplied by publisher]

https://www.ncbi.nlm.nih.gov/pubmed/29428597?dopt=Abstract

Source: Brain Research - February 8, 2018 Category: Neurology Authors: Gamal M, Moawad J, Rashid L, Attia Morcos M, Sharawy N Tags: Brain Res Source Type: research

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