WDR4 promotes HCC pathogenesis through N < sup > 7 < /sup > -methylguanosine by regulating and interacting with METTL1

CONCLUSIONS: WDR4 may contribute to HCC pathogenesis by interacting with and regulating the expression of METTL1 to synergistically modulate the m7G modification of target mRNAs in tumor cells.PMID:38493882 | DOI:10.1016/j.cellsig.2024.111145

Source: Cellular Signalling - March 17, 2024 Category: Cytology Authors: Rui Dong Chuanxu Wang Bo Tang Yayu Cheng Xuehui Peng Xiaomin Yang Bing Ni Jing Li Source Type: research