Characterization of palmitic acid toxicity induced insulin resistance in HepG2 cells

CONCLUSION: Thus, our data clearly demonstrate that both these mechanisms work simultaneously and more importantly, identified a threshold of HepG2 cells, which when crossed led to the pathological state of IR in response to PA.PMID:38431059 | DOI:10.1016/j.tiv.2024.105802
Source: Toxicology in Vitro - Category: Toxicology Authors: Source Type: research