Mir ‐200a‐3p regulates epithelial‐mesenchymal transition and inflammation in chronic rhinosinusitis with nasal polyps by targeting zeb1 via erk/p38 pathway

ConclusionsMiR-200a-3p suppresses EMT and inflammation by regulating the expression of ZEB1 via the ERK/P38 pathway. Our study provides new ideas for protecting nasal epithelial cells from tissue remodeling and finding a possible target for disease.This article is protected by copyright. All rights reserved
Source: International Forum of Allergy and Rhinology - Category: Allergy & Immunology Authors: Tags: ORIGINAL ARTICLE Source Type: research