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Specialty: Neurology
Condition: Carbon Monoxide Poisoning

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Total 264 results found since Jan 2013.

Association of Exhaled Carbon Monoxide With Stroke Incidence and Subclinical Vascular Brain Injury: Framingham Heart Study Clinical Sciences
Conclusions— In this large, community-based sample of individuals without clinical stroke/transient ischemic attack at baseline, higher exhaled CO was associated with a greater burden of subclinical cerebrovascular disease cross-sectionally and with increased risk of stroke/transient ischemic attack prospectively. Further investigation is necessary to explore the biological mechanisms linking elevated CO with stroke.
Source: Stroke - January 25, 2016 Category: Neurology Authors: Nayor, M., Enserro, D. M., Beiser, A. S., Cheng, S., DeCarli, C., Vasan, R. S., Seshadri, S. Tags: Pathophysiology, Epidemiology, Primary Prevention, Magnetic Resonance Imaging (MRI), Ischemic Stroke Clinical Sciences Source Type: research

Association Between Carbon Monoxide Intoxication and Incidence of Ischemic Stroke: A Retrospective Nested Case-Control Study in South Korea
Severe neurological sequelae occur in patients with carbon monoxide (CO) intoxication; however, whether the latter increases the long-term risk of developing ischemic stroke is unclear. We investigated the association between CO intoxication and ischemic stroke using data from the Korean National Health Information Database.
Source: Journal of Stroke and Cerebrovascular Diseases - December 3, 2020 Category: Neurology Authors: Kyeongmin Kwak, Min Kim, Won-Jun Choi, Young-Su Ju, Jong-Tae Park Source Type: research

Nutraceutical Induction and Mimicry of Heme Oxygenase Activity as a Strategy for Controlling Excitotoxicity in Brain Trauma and Ischemic Stroke: Focus on Oxidative Stress.
Authors: McCarty MF, Lerner A Abstract Introduction: Ischemic stroke and traumatic brain injury are leading causes of acute mortality, and in the longer run, major causes of significant mental and physical impairment. Most of the brain neuronal cell death in the minutes and hours following an ischemic stroke or brain trauma is mediated by the process of excitotoxicity, in which sustained elevations of extracellular glutamate, reflecting a failure of ATP-dependent mechanism which sequester glutamate in neurons and astrocytes, drive excessive activation of NMDA receptors. Areas covered: A literature search was undertake...
Source: Expert Review of Neurotherapeutics - December 9, 2020 Category: Neurology Tags: Expert Rev Neurother Source Type: research

Nutraceutical induction and mimicry of heme oxygenase activity as a strategy for controlling excitotoxicity in brain trauma and ischemic stroke: focus on oxidative stress
Expert Rev Neurother. 2021 Feb;21(2):157-168. doi: 10.1080/14737175.2021.1861940. Epub 2020 Dec 28.ABSTRACTIntroduction: Ischemic stroke and traumatic brain injury are leading causes of acute mortality, and in the longer run, major causes of significant mental and physical impairment. Most of the brain neuronal cell death in the minutes and hours following an ischemic stroke or brain trauma is mediated by the process of excitotoxicity, in which sustained elevations of extracellular glutamate, reflecting a failure of ATP-dependent mechanism which sequester glutamate in neurons and astrocytes, drive excessive activation of N...
Source: Expert Review of Neurotherapeutics - December 8, 2020 Category: Neurology Authors: Mark F McCarty Aaron Lerner Source Type: research