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Specialty: Nuclear Medicine
Condition: Heart Failure
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Total 1 results found since Jan 2013.
P53 mediates the stimulation of mitochondrial reactive oxygen species and suppression of 18F-FDG uptake and glycolysis in isoprenaline-treated H9C2 cardiomyocytes
Conclusions: These findings demonstrate that ISO treatment decreases cardiomyocyte 18F-FDG uptake by downregulating glycolytic flux and further reveals that ROS-p53 signaling plays a critical role in this metabolic response.
Source: Journal of Nuclear Medicine - May 18, 2021 Category: Nuclear Medicine Authors: Lee, J. H., Kim, D. H., Kim, M., Cho, Y. S., Jung, K.-H., LEE, K.-H. Tags: Cardiovascular Basic Science Source Type: research
