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Source: Cellular Physiology and Biochemistry
Condition: Hypertension
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Total 4 results found since Jan 2013.
SphK1/S1P Mediates PDGF-Induced Pulmonary Arterial Smooth Muscle Cell Proliferation via miR-21/BMPRII/Id1 Signaling Pathway
Conclusion: Our study indicates that SphK1/S1P pathway plays an important role in PDGF-induced PASMC proliferation via miR-21/BMPRII/Id1 axis and targeting against SphK1/S1P axis might be a novel strategy in the prevention and treatment of pulmonary arterial hypertension (PAH).Cell Physiol Biochem 2018;51:487 –500
Source: Cellular Physiology and Biochemistry - November 19, 2018 Category: Cytology Source Type: research
Calcineurin/NFAT Signaling Modulates Pulmonary Artery Smooth Muscle Cell Proliferation, Migration and Apoptosis in Monocrotaline-Induced Pulmonary Arterial Hypertension Rats
Conclusion: Our results demonstrate that CaN/NFAT signaling is activated and involved in the modulation of PASMC proliferation, migration and apoptosis in MCT-induced PAH.Cell Physiol Biochem 2018;49:172 –189
Source: Cellular Physiology and Biochemistry - August 22, 2018 Category: Cytology Source Type: research
Tetrahydroxystilbene Glycoside Improves Microvascular Endothelial Dysfunction and Ameliorates Obesity-Associated Hypertension in Obese ZDF Rats Via Inhibition of Endothelial Autophagy
Conclusion: Endothelial dysfunction in ZDF rats is partially attributable to excessive autophagy. TSG improves endothelial function and exerts hypotensive effectsvia regulation of endothelial autophagy.Cell Physiol Biochem 2017;43:293 –307
Source: Cellular Physiology and Biochemistry - August 30, 2017 Category: Cytology Source Type: research
MicroRNA-222 Promotes the Proliferation of Pulmonary Arterial Smooth Muscle Cells by Targeting P27 and TIMP3
Conclusion: miR-222 promotes PASMC proliferation at least partially through targeting P27 and TIMP3.Cell Physiol Biochem 2017;43:282 –292
Source: Cellular Physiology and Biochemistry - August 30, 2017 Category: Cytology Source Type: research
