Reduced SPOCK1 expression inhibits non-small cell lung cancer cell proliferation and migration through Wnt/ β-catenin signaling.

CONCLUSIONS: The expression of SPOCK1 served as a tumor promoter, possibly through the Wnt/β-catenin signaling pathway in NSCLC. Targeting SPOCK1 could be a potential therapeutic strategy in NSCLC. PMID: 29461591 [PubMed - in process]
Source: European Review for Medical and Pharmacological Sciences - Category: Drugs & Pharmacology Tags: Eur Rev Med Pharmacol Sci Source Type: research