S100A9 exacerbates sepsis-induced acute lung injury via the IL17-NF κB-caspase-3 signaling pathway

CONCLUSIONS: S100A9 as a key biomarker of sepsis-induced acute lung injury, and exacerbates lung damage and epithelial cell apoptosis induced by LPS via the IL-17-NFκB-caspase-3 signaling pathway.PMID:38588614 | DOI:10.1016/j.bbrc.2024.149832

Source: Biochemical and Biophysical Research communications - April 8, 2024 Category: Biochemistry Authors: Hui Pei Jianming Chen Jie Qu Zhongqiu Lu Source Type: research