A novel hepatitis B virus capsid assembly modulator QL-007 inhibits HBV replication and infection through altering capsid assembly

In conclusion, our findings underscore the potential of QL-007 as an effective agent against HBV replication and introduce it as a novel CpAM for the antiviral treatment of chronic hepatitis B.PMID:37683938 | DOI:10.1016/j.antiviral.2023.105715
Source: Antiviral Research - Category: Virology Authors: Source Type: research