Prmt5 deficiency inhibits CD4+ T-cell Klf2/S1pr1 expression and ameliorates EAE disease
Protein arginine methyltransferase 5 (Prmt5) is the main type II methyltransferase, catalyzes protein arginine residue symmetric dimethylation, and modulates normal cellular physiology and disease progression....
Source: Journal of Neuroinflammation - Category: Neurology Authors: Yingxia Zheng, Zheyi Chen, Bingqian Zhou, Shiyu Chen, Ningdai Chen and Lisong Shen Tags: Research Source Type: research