Interleukin-33 as a biomarker affected intrathecal synthesis of immunoglobulin in NMOSD and MOGAD

Conclusions: Thus, we concluded that IL-33 may induce dysfunction of the blood brain barrier and lead to intrathecal synthesis of immunoglobulin in the AQP4+NMOSD and MOGAD, especially in MOGAD. It maybe as a biomarker, at least in part, was involved in the demyelinating diseases of the central nervous system.

Source: European Neurology - April 26, 2023 Category: Neurology Source Type: research