PPAR γ mediates the anti-pulmonary fibrosis effect of icaritin

CONCLUSIONS: Icaritin has therapeutic potential against pulmonary fibrosis via the inhibition of myofibroblast differentiation, which may be mediated by PPARγ.PMID:34153405 | DOI:10.1016/j.toxlet.2021.06.014
Source: Toxicology Letters - Category: Toxicology Authors: Source Type: research